4300 | wileyonlinelibrary.com/journal/jam J Appl Microbiol. 2022;132:4300–4309. © 2022 Society for Applied Microbiology.
Received: 13 January 2022
|
Revised: 11 March 2022
|
Accepted: 21 March 2022
DOI: 10.1111/jam.15542
ORIGINAL ARTICLE
Activity of poly(methacrylic acid)-silver nanoparticles on
fluconazole-resistant Candida albicans strains: Synergistic
and cytotoxic effects
Cecília Maria Cruz Falcão
1
|
Audrey Andrade
2,3
|
Vanderlan Nogueira Holanda
4
|
Regina Celia Bressan Queiroz de Figueiredo
4
|
Eulália Azevedo Ximenes
5
|
Anderson Stevens Leonidas Gomes
1,2
1
Postgraduate Program in Dentistry,
Federal University of Pernambuco,
Recife, Brazil
2
Department of Physics, Federal
University of Pernambuco, Recife,
Brazil
3
Laboratory of Microscope and
Microanalysis, Strategic Technologies
Center of Northeast, Recife, Brazil
4
Department of Microbiology, Aggeu
Magalhaes Research Centre, Recife,
Brazil
5
Department of Antibiotics, Federal
University of Pernambuco, Recife,
Brazil
Correspondence
Cecília Maria Cruz Falcão, Postgraduate
Program in Dentistry, Federal
University of Pernambuco, 50670-901,
Recife, Prince Edward Island, Brazil.
Email: ceciliaodonto@gmail.com
Funding information
Conselho Nacional de Desenvolvimento
Científico e Tecnológico, Grant/Award
Number: PQ-1A 307259/2015-3;
Coordenação de Aperfeiçoamento de
Pessoal de Nível Superior, Grant/Award
Number: 001CAPES-UFPE; Instituto
Nacional de Fotônica, Grant/Award
Number: INCT 465763/2014-6
Abstract
Aims: To synthesize and evaluate the antifungal activity of poly(methacrylic acid)-
silver nanoparticles (PMAA-AgNPs) against nine Candida albicans isolated from
clinical specimens.
Methods and Results: The effects of PMAA-AgNPs-fluconazole combination was
analysed by checkerboard methodology. The synergistic potential of PMAA-AgNPs-
fluconazole was determined by the fractional inhibitory concentration index (FICI).
The inhibition of germ tube formation and the determination of PMAA-AgNPs cy-
totoxicity were also performed. All C. albicans strains were susceptible to PMAA-
AgNPs and resistant to fluconazole. PMAA-AgNPs at subinhibitory concentrations
restored the susceptibility of resistant C. albicans to fluconazole, whose FICI ranged
from 0.3 to 0.5. The synergistic interaction of the combination was observed in eight
of nine strains. The PMAA-AgNPs-fluconazole combination was also able to inhibit
the germ tube formation. PMAA-AgNPs showed a dose-dependent decrease in vi-
ability for cells tested, with 50% cytotoxic concentration (CC
50
) values of 6.5, 4.9 and
6.8 μg ml
−1
for macrophages, fibroblasts and Vero cells, respectively.
Conclusions: This study demonstrated that, in general, PMAA-AgNPs acts synergis-
tically in combination with fluconazole, inhibiting fluconazole-resistant C. albicans
strains. PMAA-AgNPs-fluconazole combination was also able to inhibit germ tube
formation, an important virulence factor. Inhibitory effect of PMAA-AgNPs alone or
in combination was higher in C. albicans than in mammalian cells.
Significance and Impact of Study: This study shows the potential of PMAA-AgNPs
combined with fluconazole to inhibit fluconazole-resistant C. albicans strains.
KEYWORDS
Candida albicans, fluconazole, germ tube, silver nanoparticles, synergism
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