Open Access
Mnaa et al., Adv Tech Biol Med 2015, 3:1
DOI: 10.4172/2379-1764.1000121
Open Access
Volume 3 • Issue 1 • 1000121
Adv Tech Biol Med
ISSN: 2379-1764 ATBM, an open access journal
Keywords: Black berry; White Berry; Ccl
4
; Mda; Fucosidase;
Hepatotoxic
Introduction
Liver the largest organ in the vertebrate body is the major site
of xenobiotic metabolism. Mostly, research concerned with carbon
tetrachloride CCl
4
showed infection and chemical toxic induction for
liver damage. Most remarkable pathological characteristics of CCl
4
are
fatty liver, cirrhosis and necrosis, which have been resulted from the
reactive intermediates as CCl
3
.
metabolized by the function cytochrome
p450 in the endoplasmic reticulum [1]. Blocking or retarding the
chain reaction of oxidation is one strategy to prevent or treat oxidative
stress induced hepatotoxicity. Intake of oxygen radical scavengers
involved in phytochemicals may be a good defense mechanism for
hepatoprotection. Whenever ameliorating the antioxidant status,
especially improvement of phase II detoxifying and elevation of the
antioxidant substance content surely afect on the healthy situation.
Mulberry fruits under family Moraceae are widely regarded as
a nutritious food and it can be eaten freshly or widely used in the
production of wine, fruit juice, jam and canned food [2]. Berries as well
used efectively in medical industries for treatment of sore throat, fever,
hypertension and anemia [3]. Moreover, white mulberry fruit is used to
protect against liver and kidney damage, strengthen the joints, improve
eyesight, and have antiaging efects [4]. In a chemical comparison
study, white (Morus alba L.), red (M. rubra L.) and black (M. nigra
L.) mulberry fruits investigated to grow in Turkey [5]. Tey observed
that black ones contain 1422 mg gallic acid equivalents and 276 mg
quercetin equivalents per 100 g fresh matter. On the other hand, M.
alba had the highest fat content, and ascorbic acid content.
Moreover, black berry is closely linked to its protective properties
against free radical attack induced by fuoride hepatotoxic metal.
Tey found as well that black berry decreased the hepatotoxicity
and oxidative stress of sodium fuoride (NaF) [6]. Hepatotoxicity of
NaF elevated liver thiobarbituric acid reactive substances (TBARS)
and nitric oxide (NO), while reduced superoxide dismutase (SOD),
catalase (CAT), total antioxidant capacity and glutathione (GPX).
Tey examined the increased values of plasma transaminase, creatine
kinase, lactate dehydrogenase, total lipids, cholesterol, triglycerides,
low density lipoprotein-cholesterol, and the decreased value of high
density lipoprotein-cholesterol. Tese efects have been induced by
NaF and ameliorated by black berry juice. Common traditional word in
the Middle East is saying that care is much better than cure. Sufcient
evidence has demonstrated that CCl
4
intermediates deleterious efects
in liver, however it is not clear exactly the defense mechanism for the
edible plants. Te present study tries to discover the actions if fresh
white or black berries could perform as care or cure agents against
induced hepatotoxicity in rat model.
Material and Methods
Fresh white (Morus alba L.) and black (Morus Nigra L.) mulberry
fruits were purchased from the local market (Monofa, Egypt). All
*Corresponding author: Emad Shaker, Chemistry Department, Agriculture College,
Minia University, Minia, Egypt, Tel: 201224157188; E-mail: eshaker10@hotmail.com
Received December 11, 2014; Accepted February 10, 2015; Published February
16, 2015
Citation: Mnaa S, Aniess W, Olwy Y, Shaker E (2015) Antioxidant Activity of
White (Morus alba L.) and Black (Morus nigra L.) Berries against CCl4 Hepatotoxic
Agent. Adv Tech Biol Med 3: 121. doi: 10.4172/2379-1764.1000121
Copyright: © 2015 Mnaa S, et al. This is an open-access article distributed under
the terms of the Creative Commons Attribution License, which permits unrestricted
use, distribution, and reproduction in any medium, provided the original author and
source are credited.
Abstract
Background and Aim: Our research investigates black, white and mixture of fresh berries role in liver injury
caused by CCl
4
through biochemical parameters and histopathological determinations.
Methods: Using berry as preventing or caring agents in comparing with treating or relieving or might curing
agents against CCl
4
hepatotoxicity. Injection with CCl
4
was in the second 10 days, while preventing showed that
feeding with berry on the frst 10 days and relieving was feeding with berry on the third 10 days.
Results: Preventing infected rats with white berry increased weight (17%), food effciency ratio FER (6%),
decreased aspartate aminotransferase (AST) (81%), urea (25.5%) comparing to infected rat group. Risk factor in
lipid profle showed improvement by preventing black, white followed by mixture berries (26-58%) compared with
CCl
4
group. Preventing with black, white then mixture berry showed signifcant improvement for risk factor. Generally,
optimistic data has been found for preventing black berry in malondialdehyde (MDA) and fucosidase values in the
fnal stage. In the frst stage, preventing white and mixture fresh berry showed signifcant increase for antioxidant and
fucosidase activity, respectively. Histopathological profles indicated that relieving black berry and protected mixture
were much enhanced than protected black and relieving mixture, respectively.
Conclusion: Carbon tetrachloride is showing infection and hepatotoxicity in liver diseases due to its reactive
intermediates. White, black and their equal mixture in the diet of Sprague-Dawley rats showed protective and even
relieving for CCl
4
toxicity.
Antioxidant Activity of White (Morus alba L.) and Black (Morus nigra L.)
Berries against CCl4 Hepatotoxic Agent
Said Mnaa
1
, Walaa Aniess
2
, Yaser Olwy
2
and Emad Shaker
3
*
1
Nutrition Department, Specifc Education Collage, Monofa University, Monofa
2
Nutrition Department, Specifc Education Collage, Ain-Shams University, Cairo, Egypt
3
Chemistry Department, Agriculture College, Minia University, Minia, Egypt
Advanced Techniques in
Biology & Medicine
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ISSN: 2379-1764
Research Article