Okoli et al., 2016
Biosciences Research in Today’s World | July 2015 | Volume 1 | Pages 47-54 47
Biological effect of aqueous extract of Heinsia crinita on lipid
peroxidation and angiotensin-1-converting enzyme in vitro
Esther E. Nwanna*, Ganiyu Oboh, Bukola C. Adedayo, Taiwo M. Adewuni and Isaac Ejakpovi
Functional Food and Nutraceutical Unit, Department of Biochemistry, Federal University of Technology, Akure, PMB
704, Akure 340001, Nigeria
ABSTRACT
Heinsia crinita; a common vegetable in the south-eastern part of Nigeria with the local name “Atama” have
been used as a component of various herbal portions in ethnomedicine. The plant part has been
previously used in the treatment of umbilical hernia and skin rashes. There is dearth of information on the
scientific rationale behind the use of this plant. This study was designed to investigate the
pharmacological potentials of aqueous extracts of Heinsia crinita for the management/prevention of
hypertension. Fresh, matured green leaves of Heinsia crinita were harvested from a local farm in Cross
River State, Nigeria. The total phenol, total flavonoids, 1,1-diphenyl-2-picrylhydrazyl (DPPH) scavenging
ability and ability of the extract to chelate Fe
2+
as well as inhibition of Fe
2+
-induced lipid peroxidation in rat
penile homogenate arginase and angiotensin-1-converting enzyme inhibitory activity were assessed. The
results revealed that the extract had high total phenol and total flavonoid content. The extract also
exhibited inhibitory effect on Arginase (EC50 = 3.93 mg/mL) and angiotensin-1-converting enzyme (ACE)
(EC50 = 0.77 mg/mL). Furthermore, the extract exhibited strong antioxidant capacity as typified by DPPH
scavenging and Fe
2+
chelating abilities coupled with the inhibition of Fe
2+
-induced lipid peroxidation in rat
penile homogenate in vitro. The bioactive constituents with beneficial medicinal properties coupled with
the antioxidant activities and its inhibitory effect on key enzymes linked with hypertension may be the
mechanism by which the extract manage and/or prevent hypertension and other cardiovascular diseases.
Citation: Nwanna EE, Oboh G, Adedayo BC, Adewuni TM and Ejakpovi I (2015). Biological effect of aqueous extract of Heinsia crinita
on lipid peroxidation and angiotensin-1-converting enzyme in vitro. Biosciences Research in Today’s World 1: 47-54.
doi:10.5281/zenodo.216579
Received May 28, 2015; Accepted July 2, 2015; Published July 19, 2015.
Copyright: © 2015 Nwanna et al. This is an open-access article distributed under the terms of the Creative Commons Attribution
License, which permits unrestricted use, distribution, and reproduction in any medium, provided the original author and source are
credited. BRTW is the official journal publication of BRSF.
Competing Interests: The authors have declared that no competing interests exist.
* E-mail: esthernwanna@gmail.com
This research work was presented at the Biosciences Research Support Foundation (BRSF)’s 1
st
International
Conference on Biosciences Research (ICBR), Awka, Nigeria, 25-27 May 2015.
Keywords: Heinsia crinita; ethnomedicine; arginase; hypertension.
1. INTRODUCTION
Hypertension is a multifactorial genetic-related
disease that precedes cardiovascular mortality
and morbidity in developed and developing
countries [1]. Renin produces angiotensin I from
angiotensinogen, after which it is converted to a
potent vasoconstrictor,angiotensin II, by
angiotensin-1-converting enzyme (ACE). ACE
also inactivates bradykinin,which has
vasodilating action and promotes the secretion
of aldosterone. As such, inhibition of ACE
activity will yield major antihypertension benefits
and this have been considered a useful
therapeutic approach in the management and/or
treatment of high blood pressure [2].
Dysfunction of the endothelial tissues plays a
major role in the development of erectile
dysfunction (ED) and this endothelium
dysfunction is impaired by increased oxidative
stress and inflammatory conditions [3]. Arginase
expressed in the human corpus cavernosum
tissue and which catalyse the conversion of L-
arginine to ornithine plus urea have been
implicated in ED [4] and arginase activity have
been reported to increase with hyperglycemia
and aging [5]. Increased arginase can also
provide ornithine for synthesis of polyamines via
ornithine decarboxylase (ODC) and
proline/collagen via ornithine aminotransferase
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