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Biomimetic Concealing of PLGA Microspheres in a 3D
Scaffold to Prevent Macrophage Uptake
Silvia Minardi, Bruna Corradetti, Francesca Taraballi, Monica Sandri,
Jonathan O. Martinez, Sebastian T. Powell, Anna Tampieri, Bradley K. Weiner,
and Ennio Tasciotti*
1. Introduction
Biomaterials mimicking the structure and composition of the
extracellular matrix (ECM) are capable of acting as induc-
tive templates for the formation of site-specific functional
tissues.
[1]
The reciprocal interaction between cells and ECM
is key for tissue regeneration and the consequent tissue
homeostasis.
[2]
With respect to synthetic materials, hosts
favorably respond to collagenous materials, promoting mate-
rial integration.
[3,4]
Collagen, the most abundant structural
protein in the ECM of connective tissues, plays a prominent
role in maintaining the integrity of tissue architecture and has
been successfully used in several tissue engineering applica-
tions.
[5–7]
Collagen is a highly dynamic component, providing
essential information for cell attachment,
[8]
migration,
[9]
and
organization.
[10]
However, collagen alone lacks the ability to
recapitulate the complexity of tissues’ milieu, therefore bio-
active factors are necessary additions to address any specific DOI: 10.1002/smll.201503484
Scaffolds functionalized with delivery systems for the release of growth factors
is a robust strategy to enhance tissue regeneration. However, after implantation,
macrophages infiltrate the scaffold, eventually initiating the degradation and
clearance of the delivery systems. Herein, it is hypothesized that fully embedding
the poly( D,L-lactide-co-glycolide acid) microspheres (MS) in a highly structured
collagen-based scaffold (concealing) can prevent their detection, preserving the
integrity of the payload. Confocal laser microscopy reveals that non-embedded MS
are easily internalized; when concealed, J774 and bone marrow-derived macrophages
(BMDM) cannot detect them. This is further demonstrated by flow cytometry, as
a tenfold decrease is found in the number of MS engulfed by the cells, suggesting
that collagen can cloak the MS. This correlates with the amount of nitric oxide and
tumor necrosis factor-α produced by J774 and BMDM in response to the concealed
MS, comparable to that found for non-functionalized collagen scaffolds. Finally, the
release kinetics of a reporter protein is preserved in the presence of macrophages,
only when MS are concealed. The data provide detailed strategies for fabricating
three dimensional (3D) biomimetic scaffolds able to conceal delivery systems and
preserve the therapeutic molecules for release.
Tissue Engineering
Dr. S. Minardi, Dr. B. Corradetti, Dr. F. Taraballi,
Dr. J. O. Martinez, S. T. Powell, Dr. B. K. Weiner,
Dr. E. Tasciotti
Department of Regenerative Medicine
Houston Methodist Research Institute (HMRI)
6670 Bertner Ave., Houston, TX 77030, USA
E-mail: etasciotti@HoustonMethodist.org
Dr. S. Minardi, Dr. M. Sandri, Dr. A. Tampieri
Institute of Science and Technology for Ceramics-CNR (ISTEC-CNR)
Via Granarolo 64, 48018 Faenza, RA, Italy
Dr. B. Corradetti
Department of Life and Environmental Sciences
Università Politecnica delle Marche
60131 Ancona, Italy
Dr. B. K. Weiner
Department of Orthopedic Surgery
Houston Methodist Hospital
6550 Fannin St., Houston, TX 77030, USA
small 2016, 12, No. 11, 1479–1488