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Current Pharmaceutical Biotechnology, 2012, 13, 2669-2681 2669
Nanoparticle Engineering Enhances Anticancer Efficacy of Andrographol-
ide in MCF-7 Cells and Mice Bearing EAC
Partha Roy
a
, Suvadra Das
a
, Anushree Mondal
b
, Urmi Chatterji
b
and Arup Mukherjee
a,*
a
Department of Chemical Technology, University of Calcutta, Kolkata, India;
b
Department of Zoology, University of
Calcutta, Kolkata, India
Abstract: Success in cancer chemotherapy relies on efficient delivery of anti-neoplastic drugs, with minimal side-effects
on non-cancerous cells. Nanoparticulation of prospective anti-cancer drugs, that were deemed unsuitable due to short bio-
logical half life, poor water solubility and low cellular permeability, has been hypothesized to generate superior che-
motherapeutic agents, leading to reduced non-specific action and fewer side-effects. In lieu of the above, different syn-
thetic modulations on the putative anti-cancer compound andrographolide (AG) were explored to improve its therapeutic
efficiency. Our results indicated that PLGA-nanoparticulation of andrographolide diterpenoid enhanced its anti-cancer
properties three fold. Chitosan coating of AG nanoparticles further accentuated cellular localization, induced G
1
cell cycle
arrest and increased cellular toxicity and apoptosis in MCF-7 cells. The charge modulated nanoparticles were seen to trav-
erse more efficiently through the cytoplasm and accumulate in the nucleus, thus enhancing their anti-proliferative effi-
cacy. In vivo studies confirm that the nanoparticles reduced tumor weight by 68.21% as compared to 24.7% by AG, and
increased the life span of mice infected with Ehrlich ascites carcinoma (EAC) by 78.08% as compared to 23.5% for AG
alone. This was achieved through development of slow release-type nanoparticle cargo delivery devices, and enhanced the
efficiency of AGnps for targeting cancer cells. AG nanoparticles also showed sufficient promise as safe anti-cancer drugs
since they had minimal impact on animal hematology. Hence, we successfully prepared non-toxic and delivery-efficient
andrographolide nanoparticles, and established for the first time that PLGA-nanoparticulation of andrographolide and ad-
ditional chitosan coating increased its anti-cancer efficacy in human breast cancer cells and mouse EAC model.
Keywords: Andrographolide, apoptosis, chitosan, Ehrlich Ascites Carcinoma, G
1
cell cycle arrest, human breast cancer cells,
nanoparticles.
INTRODUCTION
Till date, cancer chemotherapy remains a challenge [1],
and development of anticancer agents with low toxicity and
increased specificity of action is therefore a priority. Andro-
grapholide (AG) is a labdane diterpenoid, extracted from the
leaves of Andrographis paniculata. AG, popularly known as
the ‘King of Bitters’, is a pleiotropic agent and interacts with
a diverse range of biomolecular targets. AG is known to ex-
ert immunomodulatory effects by inhibiting NF-kB [2], and
interferes with NFAT activation and ERK1/ ERK5 phos-
phorylation in T-cells. AG is a potent inducer of mitochon-
drial-mediated apoptosis in a variety of cancer cell lines [3,
4], and alters reactive oxygen species (ROS) levels in HepG2
cell lines. Andrographis paniculata extracts have been used
in Indian and Chinese traditional medicine as an anti-
infective, hepatoprotectant and anti-inflammatory agent [5].
Anti-tumor activities of AG have also been established in
cancer models [6, 7]. Like curcumin and resveratrol, AG
modulates the Pl3K/Akt signaling pathway and induces G
1
cell cycle arrest through induction of p27 and suppression of
cyclin-dependent kinase (CDK 4). However, like most plant
terpenoids, AG has significant solubility and biopharmaceu-
tical limitations, and a short biological half life of 2 hours
*Address correspondence to this author at the Department of Chemical
Technology, University of Calcutta, 92 A. P. C. Road, Kolkata-700 009,
India; Tel: 91-33-23508387; Fax: 91-33-23519755;
E-mail: arupm1234@gmail.com
[8]. These limitations deter all therapeutic applications of
AG for which different synthetic modifications of AG were
explored to improve its therapeutic efficiency [6]. Nanopar-
ticle engineering was aimed as an appropriate solution for
specific anti-cancer applications of AG in vitro and in vivo.
In the present study we report nanoparticulation of AG
within a narrow Gaussian size range. US FDA approved bio-
polymer PLGA [poly(DL-lactide-co-glycolic acid)] was pre-
ferred because of its established biodegradability and stabili-
zation in circulation [9]. It is known that surface charges of
nanoparticles often modulate cellular internalization, particu-
larly in cancer cells [10]. Consequently, polycationic deposi-
tion on the PLGA-nanoparticles was carried out to produce
cationic nanoparticles, in order to increase cellular localiza-
tion. Finally, AG nanoparticle-induced cellular effects were
studied in MCF-7 human breast cancer cells, and addition-
ally confirmed in ME-180 human cervical cancer cell lines.
The efficacy of AG and AG nanoparticles was also estab-
lished for the first time in the mouse Ehrlich Ascites Carci-
noma (EAC) model.
MATERIALS AND METHODS
Materials
All solvents and water used were of HPLC grade
(E. Merck or Spectrochem). PLGA (50:50, MW 40,000–
75,000), chitosan (MW Medium), dialysis tubing (MW cut
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