ISBT Science Series (2018) 0, 1–9
CONGRESS REVIEW
© 2018 International Society of Blood Transfusion
DOI: 10.1111/voxs.12444
NONE TOO S.M.A.LL: the global challenge of severe malarial
anaemia and its transfusion support
Christine Cserti-Gazdewich
University Health Network/University of Toronto, Toronto, ON, Canada
Received: 4 July 2018,
revised 1 August 2018,
accepted 3 August 2018
In endemic areas, the burden of malaria and anaemia converge together upon
children, with severe malarial anaemia (SMA) accounting disproportionately for
demands on limited blood supplies. The attributable morbidity and mortality
from SMA remain high, and improved outcomes hinge in part on the timeliness,
sufficiency and safety of transfusion support. The pathogenesis of SMA is com-
plex, and depreciation kinetics (towards syndrome-defining haemoglobin levels
of <50 g/l) occur acutely or insidiously, and in relation to the parasite burden
and host response. Beyond haemolysis of parasitized erythrocytes, mechanisms of
bystander loss and reduced erythropoiesis figure prominently, with parallels to
hyperhaemolysis syndrome. Involuntarily undertransfused children with SMA in
low-income countries (LIC) differ from those adults in high-income countries
(HIC) who deliberately renounce transfusion. Despite youth-related advantages in
the power to adapt to anaemia, critical reductions in oxygen-carrying capacity
illustrate the hierarchical impacts of organ anoxia, be it in terminal cardiorespi-
ratory events or irreversible neurocognitive impairments in survivors. When
resources permit, the dynamics of restored oxygen delivery by transfusion are
particularly observable in SMA, as the triggers to transfuse are so much lower
(and the odds of corresponding lactic acidosis are so much higher) than in HICs.
Questions on the best haemotherapy approaches to SMA remain, be these in
dosage, infusion rate, component preparation or matching options; these funda-
mental concerns now transcend those related to storage duration. As a persisting
global scourge, SMA therefore keeps driving and facing the mandate to bank
life-preserving blood for those with the most to gain (and otherwise lose).
Key words: blood groups, blood safety, blood supply, immunogenetics, malaria,
transfusion medicine.
Introduction: A global view on how anaemia
and malaria converge
Malarial anaemia is an ancient scourge that remains far
from eliminated.
Anaemia affects 1Á93 billion people or 27% of the
world, with its first and third most common causes owing
to iron deficiency (63%) and malaria (4%) [1], despite
these being preventable [2]. Most (89%) of the global bur-
den is in the developing world, comprising ~81 million
malaria-attributed anaemia cases per year, and ~3 million
associated years lived with disability (YLD), the absolute
and relative numbers of which are not yet decreasing [1].
Six species of Plasmodial protozoans affect humans,
the most impactful of which is P. falciparum[3, 4] on
account of its worldwide distribution and range of red
blood cell (RBC) involvement (Table 1). These parasites
are transmitted by Anopheline mosquitoes, the paired
reaches of which are tropical, although these once
extended as far as temperate zones in the North American
Great Lakes and Northern Europe prior to 20th century
eradication [5]. Today, 3Á2 billion (or 42% of the world)
remain at risk of malaria, with 300 million infections (in
Correspondence: Christine Cserti-Gazdewich, 200 Elizabeth Street, UHN
TGH BTL 3EC-306, Toronto ON, M5G-2C4, Canada E-mail:
christine.cserti@uhn.ca
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