FEMS Microbiology Letters 112 (1993) 291-300
© 1993 Federation of European Microbiological Societies 0378-1097/93/$06.00
Published by Elsevier
291
FEMSLE 05609
Pathological activities of Yersinia ruckeri,
the Enteric Redmouth (ERM) bacterium
Jesfis L. Romalde 1 and Alicia E. Toranzo
Departmento de Microbiologla y Parasitologfa, Facultad de Biologfa, Universidad de Santiago de Compostela,
Santiago de Compostela Spain
(Received 25 June 1993; accepted 26 June 1993)
Abstract: The adherence and invasive capacities as well as the pathobiological activities exhibited by Yersinia ruckeri were
examined. Although adhesive ability was dependent on the cell-line employed, all the strains showed moderate adhesion and
invasiveness in the salmon cell-line CHSE-214. With regard to the extracellular products (ECP) all of them were strongly toxic for
fish with LD50 ranging from 2 to 9.12/~g protein per g fish. In addition, all the ECP samples showed caseinase, gelatinase, amylase,
lipase and phospholipase activities, hydrolysed esculin and displayed hemolytic activities for trout, salmon, sheep and human
erythrocytes. Heat treatment (100°C for 10 min) caused the loss of all these biological activities except the hydrolysis of gelatin. On
the other hand, SDS-PAGE analysis of the LPS and protein components of the ECP revealed variations among strains depending
on the serotype. The lack of lethal effects of the LPS present in the ECP was also demonstrated.
Key words: Yersinia ruckeri; Adherence; Invasiveness; Extracellular products; Virulence; Enteric Redmouth disease
Introduction
The Enteric Redmouth (ERM) disease caused
by Yersinia ruckeri is an acute to chronic bacter-
aemia infection affecting mainly salmonid fish all
over the world [1,2]. The first description in Spain
was in 1985 [3] and since this date, the disease
Correspondence to: A.E. Toranzo, Departamento de Microbi-
ologla y Parasitologla, Facultad de Biologla, Universidad de
Santiago de Compostela, 15706 Santiago de Compostela,
Spain.
x Present address: Baylor College of Medicine, Division of
Molecular Virology, One Baylor Plaza, Texas Medical Cen-
ter, Houston, TX 77030-3498, USA.
has been detected each year, producing signifi-
cant losses mainly in rainbow trout [4].
Despite the great number of studies on epi-
demiological and serological aspects of Y. ruck-
er/, few works focussing on the virulence mecha-
nisms of this fish pathogen have been published.
We have previously demonstrated the lack of
relationship among pathogenicity of Y. ruckeri
and classical virulence factors such as hemaggluti-
nation, hydrophobicity and other cell-surface
characteristics [5]. In this work, the toxicity for
fish as well as the biological activities of the
extracellular products (ECP) of Y. ruckeri have
been evaluated in order to understand their pos-
sible role in the development of the disease. In
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