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Zhao, et al: Aggressive therapy reduces CNO
Personal non-commercial use only. The Journal of Rheumatology Copyright © 2015. All rights reserved.
Aggressive Therapy Reduces Disease Activity without
Skeletal Damage Progression in Chronic Nonbacterial
Osteomyelitis
Yongdong Zhao, Nancy A. Chauvin, Diego Jaramillo, and Jon M. Burnham
ABSTRACT. Objective. To retrospectively assess changes in disease activity and skeletal damage in children with
chronic nonbacterial osteomyelitis (CNO) after infliximab and methotrexate, with or without
zoledronic acid or nonsteroidal antiinflammatory drug (NSAID) monotherapy, using a standardized
magnetic resonance imaging (MRI) approach.
Methods. Treatment-related changes in clinical and MRI measures from aggressive therapy and
NSAID monotherapy groups (n = 9 per group) were evaluated using nonparametric methods.
Results. Pain, physical function, physician global assessment, inflammatory markers, nonvertebral
inflammatory lesion number, and maximum bone edema score all improved significantly with
aggressive therapy (p < 0.03), whereas only the maximum soft tissue inflammation severity decreased
(p = 0.02) with NSAID monotherapy. Vertebral deformities and physeal damage did not worsen in
the aggressive therapy group but 1 in the NSAID group had worsening of growth plate damage.
Conclusion. An aggressive treatment regimen in CNO improved clinical and imaging measures of
disease activity without progression of skeletal damage. (First Release May 15 2015; J Rheumatol
2015;42:1245–51; doi:10.3899/jrheum.141138)
Key Indexing Terms:
CHRONIC NONBACTERIAL OSTEOMYELITIS METHOTREXATE
NONSTEROIDAL ANTIINFLAMMATORY DRUGS ZOLEDRONIC ACID
MAGNETIC RESONANCE IMAGING INFLIXIMAB
From the Pediatric Rheumatology Department, Seattle Children’s
Hospital, Seattle, Washington; Department of Radiology, and the Division
of Pediatric Rheumatology, The Children’s Hospital of Philadelphia,
Philadelphia, Pennsylvania, USA.
Y. Zhao, MD, PhD, acting assistant professor, Pediatric Rheumatology
Department, Seattle Children’s Hospital; N.A. Chauvin, MD, Assistant
Professor; D. Jaramillo, MD, MPH, Professor, Department of Radiology,
The Children’s Hospital of Philadelphia; J.M. Burnham, MD, MSCE,
Associate Professor, Division of Pediatric Rheumatology, The Children’s
Hospital of Philadelphia.
Address correspondence to Pediatric Rheumatology, The Children’s
Hospital of Philadelphia, 3501 Civic Center Blvd., Philadelphia,
Pennsylvania 19104, USA. E-mail: burnhams@email.chop.edu
Accepted for publication March 17, 2015.
Chronic nonbacterial osteomyelitis (CNO) is an inflam-
matory bone disease that causes skeletal inflammation and
pain, and can be complicated by functional impairment,
vertebral fractures, and limb length discrepancy. Although
treatment with nonsteroidal antiinflammatory drugs (NSAID)
relieves pain in some patients
1,2,3,4,5
, those at risk for skeletal
deformities or with persistent pain require additional therapy
with tumor necrosis factor–α inhibitors (TNFi)
1,3,6,7,8
or
bisphosphonates
4,9,10,11,12, 13,14,15,16
.
CNO treatment studies have focused on clinical, labora-
tory, and radiographic improvements. Magnetic resonance
imaging (MRI) is useful to assess active bone and soft tissue
edema, periosteal reaction, and hyperostosis, as well as
components of skeletal damage, including physeal bony bar
formation and vertebral collapse. Previous MRI-based studies
have not examined specific lesion characteristics, mainly
reporting the change in the number of lesions and qualitative
bone marrow enhancement improvement
2,7,8
.
In our practice, TNFi therapy is used for patients with
active vertebral disease or CNO-related persistent pain
despite NSAID or other nonbiologic disease-modifying
therapy. Additionally, MRI is performed at diagnosis to
identify individuals with vertebral involvement, who are
offered a single dose of bisphosphonate therapy with
zoledronic acid (ZOL), with maintenance infliximab (IFX)
therapy and concomitant methotrexate (MTX). Our retro-
spective study aimed to evaluate changes in clinical and MRI
characteristics of CNO in children treated with an aggressive
therapy including IFX and MTX, with or without ZOL, and
those treated with NSAID monotherapy. Our hypothesis was
that aggressive therapy would improve pain, physical
function, and MRI characteristics of disease activity, and
prevent progression of skeletal damage.
MATERIALS AND METHODS
Participants. Patients treated for CNO in the Division of Rheumatology at
The Children’s Hospital of Philadelphia (USA) from 2006–2013 and 2–18
years of age at diagnosis were identified. Two pediatric rheumatologists (YZ
and JMB) confirmed the CNO diagnosis if unifocal or multifocal inflam-
matory bone lesions were present without evidence of malignancy or
infection according to laboratory tests and histologic examination, if
performed
1
. Patients were included if regional or whole body MRI
(WBMRI) scans contained multiplanar fluid-sensitive sequences and were
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