Vol.:(0123456789) 1 3
Archives of Virology
https://doi.org/10.1007/s00705-018-3987-3
ORIGINAL ARTICLE
Identifcation of a conserved neutralizing epitope in the G‑protein
of Chandipura virus
Daya V. Pavitrakar
1
· Rekha G. Damle
1
· Anuradha S. Tripathy
1
· Pratip Shil
1
Received: 18 April 2018 / Accepted: 14 June 2018
© Springer-Verlag GmbH Austria, part of Springer Nature 2018
Abstract
Chandipura virus (CHPV), associated with an encephalitic illness in humans, has caused multiple outbreaks with high
mortality in central and western India in recent years. The present study compares surface glycoprotein (G-protein) from
prototype and recent outbreak strains using in silico tools and in vitro experiments. In silico epitope predictions (B-cell and
T-helper cell) for the sequences, 3D structure prediction and comparison of the G-proteins of the strains: I653514 (Year
1965), CIN0327 (Year 2003) and 148974 (Year 2014) revealed that the CHPV G-protein is stable and antigenic determinants
are conserved. A monoclonal antibody developed against strain CIN0327 (named NAbC) was found to neutralize prototype
I653514 as well as the currently circulating strain 148974. In silico antigen-antibody interaction studies using molecular
docking of predicted structures of NAbC and G-proteins of various CHPV strains led to the identifcation of a conserved
neutralizing epitope in the fusion domain of G-protein, which also contained a putative T-helper peptide. The identifcation
of a conserved neutralizing epitope in domain IV (fusion domain amino acids 53 to 172) of CHPV G-protein is an important
fnding that may have the scope towards the development of protective targets against CHPV infection.
Introduction
Taxonomically, Chandipura virus (CHPV) belongs to the
Vesiculovirus genus of the Rhabdoviridae family. CHPV
causes acute encephalitis in human paediatric populations
[1]. This virus was responsible for encephalitis outbreaks
reported in Andhra Pradesh/Telengana and Maharashtra,
India during the years 2003 and 2004 respectively [1, 2]. It is
a negative sense, single stranded RNA virus which consists
of fve structural proteins in the canonical order N-P-M-G-
L: a nucleoprotein (N), a nucleocapsid-associated phospho-
protein (P), a matrix protein (M), a glycoprotein (G) and the
viral polymerase (RdRp, L) located between 3’ leader and
5’ trailer sequences.
The G-protein is a trimeric trans-membrane glycoprotein
that enables virus adsorption, assembly, budding and also
elicits an antibody response thus acting as a major antigenic
determinant [3]. Depending on the state of infection it adopts
diferent reversible conformational states: i) native state pre-
sent on the virus surface and stable above pH 7.0 [4] ii)
activated state that fuses with the target membrane [5] iii)
A fusion inactive post-fusion state which is stable under low
pH conditions [6]. In earlier studies, the structure of a pre-
fusion form of vesicular stomatitis virus (VSV) G-protein
was analysed by the molecular replacement of diferent
domains. The G-protein was divided into diferent domains,
domain I: Lateral domain (1 to 17 and 310 to 382), domain
II: Trimerization domain (18 to 35, 259 to 309, and 383 to
405), domain III: PH domain (36 to 46 and 181 to 258),
domain IV: Fusion domain (53 to 172), Cter C-terminal part
(406 to 413) and RbI-II domain: Rigid block (1 to 25 and
273 to 382) [7].
Being the major antigenic determinant, the G-protein
of CHPV requires in depth studies to better understand its
potential as a target for diferent preventive and diagnostic
approaches. The emergence of massive CHPV outbreaks in
2003 with case fatality rates as high as 56-75 % [2] as well
as isolated outbreaks thereafter from newer geographical
Handling Editor: William G Dundon.
Electronic supplementary material The online version of this
article (https://doi.org/10.1007/s00705-018-3987-3) contains
supplementary material, which is available to authorized users.
* Pratip Shil
shil.p@gov.in; shilpratip@gmail.com
1
ICMR-National Institute of Virology, 130/1 Sus Road,
Pashan, Pune 411021, India