Metoprolol oxidation polymorphism in Brazilian elderly
cardiac patients
Daniel Valente Neves
a
, Vera Lucia Lanchote
a
, Luiz de Souza
b
, Miyeko Hayashida
c
,
Maria Sueli Nogueira
c
, Natália Valadares de Moraes
a
and Evandro José Cesarino
a
*
a
School of Pharmaceutical Sciences of Ribeirão Preto,
b
School of Medicine of Ribeirão Preto, Department of Child Care and Pediatrics, and
c
College
of Nursing, Department of General and Specialized Nursing, University of São Paulo, Ribeirão Preto, São Paulo, Brazil
Keywords
CYP2D6; elderly; metoprolol; polymorphism;
phenotype
*Correspondence
Evandro José Cesarino, University of Sao
Paulo, School of Pharmaceutical Sciences of
Ribeirão Preto, Department of Clinical Analy-
sis, Toxicological and Bromatological, Avenue
of Café s/n, Campus of University of Sao
Paulo, Ribeirão Preto, São Paulo ZIP 14040-
903, Brazil.
E-mail: cesarino@fcfrp.usp.br
e-mail address: Daniel Valente Neves:
danvn83@fcfrp.usp.br; Vera Lucia Lanchote:
lanchote@fcfrp.usp.br; Luiz de Souza:
ldsouza@fmrp.usp.br; Miyeko Hayashida:
miyeko@eerp.usp.br; Maria Sueli Nogueira:
msnog@eerp.usp.br; Natália Valadares de
Moraes: nataliavaladaresdemoraes@gmail.com
Received November 16, 2012
Accepted June 15, 2013
doi: 10.1111/jphp.12109
Abstract
Objectives The purpose of this study was to phenotype the CYP2D6 in elderly
with heart disease classified as extensive metabolizer or poor metabolizers (PM) of
metoprolol, develop and validate the method of analysis of metoprolol tartrate
and its metabolite in urine using HPLC, and identify potential correlations
between anthropometric factors with metabolic ratios of metoprolol/a-OH meto-
prolol in urine.
Methods The sample was composed of 130 elderly individuals with a previously
identified type of heart condition, with normal renal and hepatic functions. The
urine of all the patients were collected 0–8 h after the administration of a pill
of 100 mg of metoprolol to determine concentrations of metoprolol and
a-hydroxymetoprolol. Those patients presenting a metabolic ratio greater than
12.6 were phenotyped as PM.
Key findings The median age of patients was 71.0 years, with a minimum of 60
and maximum of 93 years old. Three patients (2.3%) were phenotyped as PM of
metoprolol different from the rate (7–10%) of PM existing in the Caucasian
population.
Conclusions Most of the studied individuals were women, and the proportion of
elderly with heart disease classified as PM was smaller than what is usually found
among Caucasian populations.
Introduction
CYP2D6 is involved in the oxidative metabolism of
approximately 25% of all the prescribed drugs, and it
is characterized by a high rate of interindividual and
interethnic variability.
[1]
Individuals are classified into four
phenotypes according to the CYP2D6 activity: ultra-rapid
metabolizers (UMs), extensive metabolizers (EMs),
intermediate metabolizers (IMs) and poor metabolizers
(PMs).
[2,3]
Because of this polymorphism, the drug may
present a difference of 30–40 times in clearance. Hence,
the drug’s plasma concentration may be outside the thera-
peutic range and either cause adverse effects or therapeutic
failure.
[1]
Physiological changes and cardiovascular diseases in
elderly patients can affect the pharmacokinetics of certain
drugs. Prescott et al. reported that a half-life of lignocaine
was three times greater in patients with acute myocardial
infarction (AMI) when compared with healthy individu-
als.
[4]
Van Griensven et al. showed an increase in the plasma
concentrations of saruplase in patients with AMI that was
mainly due to the decreased hepatic blood flow, which
affects the metabolism of this drug.
[5]
Hanson et al. also
demonstrated that patients with chronic heart failure New
York Heart Association (NYHA) class III and IV, treated
with a single dose of 50 mg of hydralazin, presented
higher area under the curve (AUC) values compared with
patients with essential hypertension.
[6]
Orlando et al. in turn
reported a significant increase in AUC and Cmax, and a
decrease in CL/F in fluvoxamine in healthy older adults in
comparison with healthy younger adults, although no sig-
nificant differences were found in AUC, Cmax and CL/F
And Pharmacology
Journal of Pharmacy
Research Paper
© 2013 University of Sao Paulo. JPP © 2013
Royal Pharmaceutical Society, Journal of Pharmacy and Pharmacology, 65, pp. 1347–1353 1347
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