AJR:179, September 2002 759
Original Report
OBJECTIVE. The records of 10 patients with focal nodular hyperplasia inducing intrahe-
patic vein obstruction were reviewed. The purpose of this study was to describe and empha-
size the imaging features of these findings.
CONCLUSION. Focal nodular hyperplasia may be responsible for hepatic vein obstruc-
tion with hepatic vein collaterals. The relatively large size and central location of the lesions
seem to play important roles in the obstruction of the hepatic veins.
ocal nodular hyperplasia is the sec-
ond most common benign liver le-
sion after hemangioma and accounts
for 8% of all primary hepatic tumors [1, 2]. It
typically affects women of childbearing age. The
exact pathogenesis of focal nodular hyperplasia
is not known, but it seems to be the result of a
hyperplastic, rather than a neoplastic, process.
Vascular malformation or injury has been sug-
gested as the triggering mechanism of hepato-
cellular hyperplasia [1, 3, 4]. Focal nodular
hyperplasia is classically described as a nodular,
hypervascular homogenous lesion with a central
stellate scar containing malformed vascular
structures with radiating fibrous septa [1]. The
only radiologic finding may be a subtle mass ef-
fect with compression of adjacent structures,
which has been previously described in large fo-
cal nodular hyperplasia [2]. However, vascular
compression, especially hepatic vein obstruc-
tion, remains rare; to our knowledge, only one
case has been reported in the literature [5]. We
describe the clinical findings of 10 patients with
focal nodular hyperplasia resulting in hepatic
vein obstruction and compare all findings with
those of a series of 64 patients with focal nodular
hyperplasia seen during the same period.
Materials and Methods
Patients
We retrospectively reviewed the records of 74
patients with focal nodular hyperplasia from 1995
to 2000. All patients were women between 18 and
70 years old (mean age, 38 years). We classified
the records into a control group (n = 64) and a
group having focal nodular hyperplasia with he-
patic vein obstruction (compressive group) (n =
10). All patients had undergone sonography, CT,
or contrast-enhanced MR imaging of the liver, or
all three examinations. Helical CT was performed
in all patients with a triphasic dynamic exploration
during the arterial, portal, and delayed phases after
injection of iodine contrast medium. All MR im-
aging included fast T2-weighted sequences and
T1-weighted dynamic gradient-echo sequences af-
ter IV injection of gadopentetate dimeglumine.
Image Interpretation
Two radiologists reviewed all images from the
two groups in consensus. Diagnosis of focal nodu-
lar hyperplasia was made in the presence of a typi-
cal appearance on CT or MR imaging and by
histologic sampling in atypical cases.
A lesion was considered to have typical appear-
ance if it was homogeneous, was unencapsulated,
and showed regular or lobulated margins on sonog-
raphy, CT, or MR imaging. On CT, a typical lesion
was slightly hypoattenuating or isoattenuating and
enhanced on the arterial phase after injection of
contrast medium, except for a central hypoattenuat-
ing scar that later enhanced [6, 7]. On MR imaging,
a typical lesion was isointense or nearly isointense
on T1- and T2-weighted imaging and showed a
central scar that enhanced on delayed gadolinium-
enhanced T1- weighted sequences [2].
The number, size, and location of the lesions
were assessed. Focal nodular hyperplasia was clas-
Anne-Sophie Rangheard
1
Valérie Vilgrain
1
Pascale Audet
1
Dermot O’Toole
2
Marie-Pierre Vullierme
1
Dominique Valla
3
Jacques Belghiti
4
Yves Menu
1
Received June 11, 2001; accepted after revision
February 26, 2002.
1
Department of Radiology, Hospital Beaujon, 100 ave.
du Général Leclerc, 92110 Clichy, France. Address
correspondence to A.-S. Rangheard.
2
Department of Gastro-Enterology, Hospital Beaujon,
92110 Clichy, France.
3
Department of Hepatology, Hospital Beaujon,
92110 Clichy, France.
4
Department of Visceral Surgery, Hospital Beaujon,
92110 Clichy, France.
AJR 2002;179:759–762
0361–803X/02/1793–759
© American Roentgen Ray Society
F
Focal Nodular Hyperplasia Inducing
Hepatic Vein Obstruction
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