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Effects of energy balance on postprandial triacylglycerol
metabolism
Maria Maraki and Labros S. Sidossis
Introduction
Elevated levels of plasma triacylglycerols (TAG) are
associated with increased risk for atherosclerosis inde-
pendently of other known cardiovascular risk factors [1];
therefore, interventions that decrease or prevent an
increase in plasma TAG concentrations may be valuable
in reducing the risk of coronary heart disease (CHD).
Considering that free-living humans spent most of their
time in the postprandial state, along with recent evidence
of the independence of postprandial TAG as a CHD risk
factor [2], suggests that interventions should focus on the
improvement of postprandial TAG metabolism.
The effects of negative energy balance on postprandial
TAG metabolism have not been elucidated. Most of the
data come from studies investigating the effects of
an increase in energy expenditure (exercise) and/or of
reduction in energy intake (diet), either in the short-term
(acute effects), or in the long-term (accompanying with
weight loss). One of the main assumptions underlying the
hypotheses of hypotriacylglycerolemic effect of energy
deficit is that the body may intensify the clearance of
TAG from the circulation when energy is available (i.e.,
during the postprandial period) in order to replenish the
energy stores used during energy deficit.
Effects of acute negative energy balance on
postprandial triacylglycerol metabolism
Accumulated evidence suggests that a single bout of
aerobic exercise reduces postprandial TAG concentrations
(pTAG) the next day (Tables 1 and 2). However, in most of
these studies, the extra energy expenditure due to exercise
relative to rest period (net exercise energy expenditure)
has not been compensated with increase in energy intake;
therefore patients in these studies were in energy deficit
due to increased energy expenditure. Moreover, regular
physical activity does not influence pTAG when the
effects of last exercise bout, and hence of exercise-induced
Department of Internal Medicine, Sealy Center on
Aging, Institute for Translational Sciences and Shriners
Burns Institute, University of Texas Medical Branch at
Galveston, Texas, USA and Laboratory of Nutrition &
Clinical Dietetics, Department of Nutrition and
Dietetics, Harokopio University, Athens, Greece
Correspondence to Labros S. Sidossis, PhD,
Laboratory of Nutrition & Clinical Dietetics, Department
of Nutrition and Dietetics, Harokopio University,
70 El. Venizelou Ave., Athens 176-71, Greece
Tel: +30 210 954 9154; fax: +30 210 954 9141;
e-mail: lsidossis@hua.gr
Current Opinion in Clinical Nutrition and
Metabolic Care 2010, 13:608–617
Purpose of review
To present the effect of negative energy balance on postprandial triacylglycerol
concentrations (pTAG), an independent risk factor for the development of
cardiovascular disease.
Recent findings
Aerobic exercise reduces pTAG; however, recent findings confirm that this effect is only
evident with an accompanying energy deficit. Moreover, a recent study showed that
acute diet-induced energy deficit also reduces pTAG. The extent of energy deficit
required to significantly attenuate pTAG depends on the type of given meal, the type of
deficit (aerobic/resistance exercise, diet, or combination of diet and exercise), and
patients’ health status. Apart from the acute effects, prolonged energy deficit leading to
moderate weight loss attenuates pTAG, when it is combined with other known
hypotriacylglycerolemic agents, such as carbohydrate restriction.
Summary
For healthy population, it seems that it is up to patient’s preference and ability
which type of energy deficit will follow to attenuate pTAG; an energy deficit of
approximately 30 kJ/kg of body mass is required; for resistance exercise a smaller deficit
is probably sufficient. More studies are needed to investigate dose–response/plateau
effects, the effects of energy deficit–energy surplus every other day, and the threshold
of energy deficit-weight loss in diabetics and other high-risk populations. Finally,
investigation of the underlying mechanisms may be clinicall helpful in individualizing the
appropriate intervention.
Keywords
diet, energy deficit, exercise, postprandial lipemia, weight loss
Curr Opin Clin Nutr Metab Care 13:608–617
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1363-1950
1363-1950 ß 2010 Wolters Kluwer Health | Lippincott Williams & Wilkins DOI:10.1097/MCO.0b013e32833f1aae