REDUCED SALICYLIC ACID BINDING FOLLOWING
NOISE: POSSIBLE EVIDENCE FOR PRESTIN
DISRUPTION
Cahtia Adelman,
1
Jeffrey M. Weinberger
2
and Haim Sohmer
3
'Speech & Hearing Center, Hadassah University Hospital,
Jerusalem;
2
Dept. of Otolaryngology and Head & Neck Surgery,
Hadassah University Hospital, Jerusalem;
3
Dept. of Physiology;
Institute for Medical Research - Israel-Canada, Hebrew University-
Hadassah Medical School, Jerusalem
ABSTRACT
To gain insight into the mechanism of noise induced permanent threshold
shift (PTS), the magnitude of the auditory threshold elevation induced by
injection of salicylic acid (which competitively binds with the motor protein
prestin) to animals with a pre-existing PTS was compared to that in control
animals (not exposed to noise). Normal mice were exposed to a noise intensity
and duration which causes a small PTS. After determining the degree of the
resulting PTS two weeks following the noise, salicylic acid was injected. The
salicylic acid induced an additional threshold elevation and its magnitude was
compared to that in control mice which had not been noise exposed. The mean
noise induced PTS in the experimental (noise exposed) group was 25.5 dB.
Following the administration of salicylic acid to these animals, there was an
additional (salicylic acid induced) mean threshold elevation of 17.5 dB, and this
was significantly smaller than that in control (not noise exposed) mice (36.8
dB). This may be evidence for a reduced number of salicylic acid binding sites
on prestin and therefore the PTS may be due to disruption of prestin by the free
radicals produced during the noise exposure.
Correspondence: Prof. Haim Sohmer, Dept. of Physiology, Hebrew University-
Hadassah Medical School, POB # 12272, Jerusalem 91120, Israel; Tel:-972-2-
6758385; Fax:-972-26439736; e-mail: haims@ekmd.huji.ac.il
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