https://doi.org/10.1177/1120672120977343
European Journal of Ophthalmology
1–6
© The Author(s) 2020
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DOI: 10.1177/1120672120977343
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Introduction
Knobloch syndrome is a rare autosomal recessive disease,
first described by Knobloch as the association between
encephalocele and retinal detachment (RD) in a single
family.
1
Since the first description in 1971, a few publica-
tions have described additional ocular manifestations and
different severity of this syndrome. The ocular phenotype
can be variable and includes both posterior and anterior
segment manifestations such as high myopia, lens sublux-
ation and early-onset cataracts, vitreoretinal degeneration,
RD, and smooth irides.
2–4
Retinal degeneration with severe
cone-rod dystrophy (CRD) confirmed by electroretino-
gram (ERG) is another recently described manifestation
among patients with Knobloch syndrome.
4
Non ophthalmic manifestations are typical as well and
include occipital changes (cutis aplasia, encephalocele,
bone defects), brain defects (Dandy-Walker Malformation
Variable phenotype of Knobloch
syndrome due to biallelic COL18A1
mutations in children
Nadav Levinger
1
, Karen Hendler
1
, Eyal Banin
1
, Mor Hanany
1
,
Adva Kimchi
1,2
, Hadas Mechoulam
1
, Vardiella Meiner
2
,
Yoav Parag
3
, Dror Sharon
1
, Michal Macarov
1,2
and Claudia Yahalom
1
Abstract
Purpose: Knobloch syndrome is a rare, recessively inherited disorder classically characterized by high myopia, retinal
detachment, and occipital encephalocele. Our aim is to report the clinical and genetic findings of four Israeli children
affected by Knobloch syndrome.
Methods: Retrospective study of four patients diagnosed with Knobloch syndrome, who underwent full ophthalmic
examination, electroretinography, and neuroradiologic imaging. Genetic analysis included whole exome sequencing
(WES) and Sanger sequencing.
Results: The four patients included in this study had high myopia and nystagmus at presentation. Ocular findings included
vitreous syneresis, macular atrophy, macular coloboma, and retinal detachment. One child had iris transillumination
defects and an albinotic fundus, initially leading to an erroneous clinical diagnosis of albinism. Electroretinography revealed
a marked cone-rod pattern of dysfunction in all four children. Brain imaging demonstrated none to severe occipital
pathology. Cutaneous scalp changes were present in three patients. WES analysis, confirmed by Sanger sequencing
revealed COL18A1 biallelic null mutations in all affected individuals, consistent with autosomal recessive inheritance.
Conclusions: This report describes variable features in patients with Knobloch syndrome, including marked lack of eye
pigment similar to albinism in one child, macular coloboma in two children as well as advanced cone-rod dysfunction in
all children. One patient had normal neuroradiologic findings, emphasizing that some affected individuals have isolated
ocular disease. Awareness of this syndrome, with its variable phenotype may aid early diagnosis, monitoring for potential
complications, and providing appropriate genetic counseling.
Keywords
Genetics, retinal degenerations associated with systemic disease, retina, inner retinal/vitreoretinal dystrophies, genetic
disease/congenital abnormalities, pediatric ophthalmology, molecular
1
Department of Ophthalmology, Hadassah University Medical Center,
Faculty of Medicine, Hebrew University, Jerusalem, Israel
2
Department of Genetics, Hadassah-Hebrew University Medical
Center, Jerusalem, Israel
3
Department of Diagnostic Radiology, Hadassah-Hebrew University
Medical Center, Jerusalem, Israel
Corresponding author:
Claudia Yahalom, Department of Ophthalmology, Hadassah Hebrew
University Medical Center, Kiryat Hadassah POB 12000 Hadassah
University Hospital, Jerusalem 9112001, Israel.
Email: kloudia@hadassah.org.il
977343EJO 0 0 10.1177/1120672120977343European Journal of OphthalmologyLevinger et al.
research-article 2020
Original research article