Vol.:(0123456789) 1 3
Theoretical and Applied Genetics
https://doi.org/10.1007/s00122-020-03654-5
ORIGINAL ARTICLE
A durum wheat adult plant stripe rust resistance QTL and its
relationship with the bread wheat Yr80 locus
Hongyu Li
1,3
· Harbans Bariana
2
· Davinder Singh
2
· Lianquan Zhang
3
· Shannon Dillon
1
· Alex Whan
1
·
Urmil Bansal
2
· Michael Aylife
1
Received: 1 April 2020 / Accepted: 8 July 2020
© Springer-Verlag GmbH Germany, part of Springer Nature 2020
Abstract
Key message A stripe rust resistance QTL in durum wheat maps near the bread wheat Yr80 locus with the latter
reduced to 15 candidate genes.
Abstract Some wheat adult plant resistance (APR) genes provide partial resistance in the later stages of plant develop-
ment to rust diseases and are an important component in protecting wheat crops from these fungal pathogens. These genes
provide protection in both bread wheat and durum wheat. Here, we have mapped APR to wheat stripe rust, caused by the
fungal pathogen Puccinia striiformis f. sp. tritici, in a cross between durum cultivars Stewart and Bansi. Two resistance
QTLs derived from the Stewart parent were identifed in multi-generational feld trials. One QTL is located on chromosome
1BL and maps to the previously identifed Yr29/Lr46/Sr58/Pm39 multi-pathogen APR locus. The second locus, located on
chromosome 3BL, maps near the recently described bread wheat APR gene, Yr80. Fine mapping in durum and bread wheat
families shows that the durum 3BL locus and Yr80 are closely located, with the later APR gene reduced to 15 candidate
genes present in the Chinese Spring genome sequence. Distorted segregation of the durum 3BL region was observed with
the Stewart locus preferentially transmitted through pollen when compared with the equivalent Bansi region.
Introduction
Plant diseases remain a continued constraint to agricultural
crop production. Currently, one of the most signifcant dis-
ease threats to both bread wheat (Triticum aestivum L.) and
durum wheat (T. turgidum spp. durum (Desf) Husn.) is stripe
rust caused by the fungal pathogen Puccinia striiformis f. sp.
tritici, Eriks. and Henn. (Pst). More aggressive strains of
Pst with adaptation to high temperature have emerged and
expanded into new environments threatening global wheat
production (Milus et al. 2009).
Genetic resistance remains the most economically and
environmentally sustainable approach for controlling
wheat stripe rust. Two types of Pst resistance mechanisms
have been identifed in wheat, all stage resistance (ASR)
and adult plant resistance (APR). More than 80 stripe rust
resistance genes have been catalogued in bread and durum
wheats (Gessese et al. 2019). Recently, several ASR genes
(Yr5, Yr7, YrSp) have been cloned (Marchal et al. 2018) that
encode nucleotide-binding site leucine-rich repeat proteins
(NLR), a ubiquitous class of plant disease resistance proteins
found in all plant species and which provide resistance to a
diverse range of microbial pathogens and insect parasites.
NLR genes function by the direct or indirect recogni-
tion of pathogen molecules (efectors) that are introduced
into plant cells to promote pathogenesis. As yet no efec-
tor molecules that are recognised by a cognate cloned NLR
gene have been identifed in the Pst-wheat pathosystem, but
similar NLR/efector gene pairs have recently been identifed
in the closely related wheat stem rust pathosystem caused
Communicated by Hermann Buerstmayr.
Electronic supplementary material The online version of this
article (https://doi.org/10.1007/s00122-020-03654-5) contains
supplementary material, which is available to authorized users.
* Michael Aylife
Michael.aylife@csiro.au
1
CSIRO Agriculture and Food, Box 1700, Clunies Ross
Street, Canberra, ACT, Australia
2
School of Life and Environmental Sciences, Faculty
of Science, The University of Sydney Plant Breeding
Institute, 107 Cobbitty Road, Cobbitty, NSW 2570, Australia
3
Triticeae Research Institute, Sichuan Agricultural University,
211 Huimin Road, Wenjiang, Chengdu 611130, Sichuan,
China