Vol. 119, No. 2, 1984
March 15, 1984
BIOCHEMICAL AND BIOPHYSICAL RESEARCH COMMUNICATIONS
Pages 561-566
REGULATION OF THE CELLULAR ACTIN LEVELS IN RESPONSE TO
CHANGES IN THE CELL DENSITY IN ATAXIA TELANGIECTASIA
LYMPHOBLASTOID CELLS
Peter J. McKinnon and Leigh A. Burgoyne
School of Biological Sciences, Flinders University of
South Australia, Bedford Park, South Australia 5042
Received January 25, 1984
Summary: The level of actJn was found to decrease markedly when ataxia
telangiectasia lymphoblastoid cells were stepped from low to high density
culture conditions. Additionally, as the actin levels decreased the levels
of a protein species of 37K dalLons increased by orders of magnitude.
Partial proteolytic digestion of the 37K protein and actin revealed that the
primary structures of these proteins were not related, This phenomena was
observed in three out of four ata×ia telangiectasia lymphoblastoid cell
lines but not in lymphoblastoid cells derived from normal individuals.
One of the most abundant cellular proteins in eukaryotes is actin. Actin
genes are known to be members of a multigene family, and inLracellular actin
found in nonmuscle cells consists of a heterogeneous population of actin
molecules (i, 2). Six different actin molecules have been identified in
mammals; B and y actin are cytoplasmic and ~ actin is found in muscle. The
muscle actin can be further classified into four different subtypes (3).
Polymeric actin is found as the main constituent of microfilaments, which
are involved in a number of cellular functions including endocytosis,
receptor internalisation and cell motility (4). A large number of cellular
proteins have been shown to be able to bind to, and regulate the
polymerisation and depolymerisation of actin (5, 6).
This paper describes an unusual regulation of actin levels observed in
lymphoblastoid cell lines derived from patients with the disorder ataxia
telangiectasia (AT), which is characterized by ionising radiation
sensitivity and developmental abnormalities.
MATERIALS AND METHODS
Cell culture: LymphoblasLoid cell lines were obtained by Epstein Barr
Virus (EBV) transformation. Control lines were C5ABR, CIOABR and the AT
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