IP International Journal of Medical Paediatrics and Oncology 2022;8(4):165–169
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IP International Journal of Medical Paediatrics and
Oncology
Journal homepage: https://www.ijmpo.com/
Case Report
Hypohidrotic hereditary ectodermal dysplasia: A case report
Dimple Padawe
1
, Ankita Khade
1
, Vilas Takate
1
, Kishore Dighe
1
,
Sanpreet Singh Sachdev
2,
*
1
Government Dental College and Hospital, Mumbai, Maharashtra, India
2
Dept. of Oral Pathology and Microbiology, Government Dental College and Hospital, Mumbai, Maharashtra, India
ARTICLE INFO
Article history:
Received 15-11-2022
Accepted 19-12-2022
Available online 11-01-2023
Keywords:
Developmental Disorders
Genodermatoses
Hypodontia
Oligodontia
Oral Rehabilitation
ABSTRACT
Hypohidrotic Ectodermal Dysplasia (HED) is characterized by a triad of hypotrichosis (sparse hair),
hypodontia (multiple missing teeth), and hypohidrosis (inability to sweat adequately). Consequently,
patients’ quality of life is severely affected due to impairments in mastication, speech, and esthetics. Timely
oral rehabilitation of patients with HED can enable them to lead a normal life by helping them perform their
functions adequately and prevent the psychological ill-effects; in which dental professionals play a key role.
The present case report describes the oral rehabilitation and management of a 7-year-old male patient with
HED.
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1. Introduction
Ectodermal dysplasias (EDs) are a heterogeneous group
of hereditary disorders characterized by certain shared
structural and functional abnormalities in tissues derived
from the ectoderm. The ectoderm gives rise to various
structures such as hair, teeth, nails, and the central
nervous and peripheral nervous systems. Various glands
including the sweat glands, sebaceous eccrine, mammary,
and pituitary also arise from the ectoderm.
1
As the name
implies, abnormalities in the embryogenesis of the ectoderm
give rise to defective structure and function of these
derivatives which collectively constitute the spectrum of
ED.
Mutations in ectodysplasin-related genes (EDA1, EDAR,
EDARADD) and the WNT-10A gene are responsible for
ectodermal defects ED.
2
The number of alleles affected and
the different extent of involvement of the various derivative
structures give rise to numerous varieties of ED, of which
* Corresponding author.
E-mail address: sunpreetss@yahoo.in (S. S. Sachdev).
117 were identified by Freire-Maia in the 1970s.
3
Even with
all the types combined, the incidence of ED is as low as 1
out of 1,00,000 live births, and therefore, it is considered a
rare condition. Hidrotic Ectodermal Dysplasia (Clouston’s
syndrome) and Hypohidrotic Ectodermal Dysplasia (HED)
also known as Christ-Siemens-Touraine Syndrome are the
most frequent of all the types.
Records of the earliest cases of ED date back to 1792,
although the first case was described by Thurman in 1848.
4
The term Hereditary ED was suggested by Weech in 1929,
and because of the inability of the patients to perspire, he
also added the adjective anhidrotic.
5
Since all the affected
patients are never completely devoid of sweat glands, the
adjective ‘hypohidrotic’ was discerned to be more apt by
Felsher in 1944.
3
While the syndrome only affects males due to its X-
linked recessive pattern of inheritance, carrier females may
display some of the characteristics of the disease.
6
True
hypodontia due to agenesis of teeth, along with atrophy of
the maxillary and mandibular alveolar bone are classically
present in children with ED. Consequently, patients’
https://doi.org/10.18231/j.ijmpo.2022.039
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