Volume 7 Issue 4 • 1000358
J Alzheimers Dis Parkinsonism, an open access journal
ISSN:2161-0460
Open Access Research Article
Sugimoto et al., J Alzheimers Dis Parkinsonism 2017, 7:4
DOI: 10.4172/2161-0460.1000358
Journal of
Alzheimer’s Disease & Parkinsonism
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ISSN: 2161-0460
*Corresponding author: Hiwasa T, Department of Biochemistry and Genetics,
Chiba University, Graduate School of Medicine, Japan, Tel: 81432262541; E-mail:
hiwasa_takaki@faculty.chiba-u.jp
Received July 14, 2017; Accepted July 27, 2017; Published August 03, 2017
Citation: Sugimoto K, Tomiyoshi G, Mori M, Kuwabara S, Hirano S, et al. (2017)
Identifcation of Serum Anti-GADD34 Antibody as a Common Marker of Diabetes
Mellitus and Parkinson Disease. J Alzheimers Dis Parkinsonism 7: 358. doi:
10.4172/2161-0460.1000358
Copyright: © 2017 Sugimoto K, et al. This is an open-access article distributed
under the terms of the Creative Commons Attribution License, which permits
unrestricted use, distribution, and reproduction in any medium, provided the original
author and source are credited.
Keywords: GADD34; PPP1R15A; Parkinson disease; Diabetes
mellitus; Serum antibody biomarker
Abbreviations: ABI, ankle-branchial index; aCI, acute-phase cerebral
infarction; ALB, albumin; AlphaLISA, amplifed luminescence proximity
homogeneous assay-linked immunosorbent assay; ALS, amyotrophic
lateral sclerosis; AST, aspartate aminotransferase; ATF4, activating
transcription factor 4; Atg12, autophagy 12; ATP2B4, ATPase, Ca
++
-
transporting, plasma membrane 4; AUC, area under the ROC curve;
BMI, body mass index; BMP-1, bone morphogenetic protein 1; CAVI,
cardio-ankle vascular index; CHOP, C/EBP homologous protein; CI,
confdence interval; CKD, chronic kidney disease; CRE, creatinine; CVD,
cardiovascular disease; DHPS, deoxyhypusine synthase; DM, diabetes
mellitus; ECSA, esophageal carcinoma SEREX antigen; ER, endoplasmic
reticulum; E. coli, Escherichia coli; GAD65, 65kDa form of glutamic acid
decarboxylase; GADD3, growth arrest and DNA-damage-inducible gene
34; GST, glutathione-S-transferase; Hb, hemoglobin; HbA1c, glycated
hemoglobin; HD, healthy donor; HDL-C, high-density lipoprotein
cholesterol; Hsp, heat shock protein; IA-2, insulinoma antigen 2; LDL-C,
low-density lipoprotein cholesterol; LRRK2, leucine- rich repeat kinase
2; max IMT, maximum intima-media thickness; PD, Parkinson disease;
PERK, protein kinase RNA-like endoplasmic reticulum kinase; PINK1,
PTEN-induced putative kinase 1; PRKN, parkin RBR E3 ubiquitin protein
ligase; ROC, receiver operating curve; RPA2, replication protein A2;
SD, standard deviation; SEREX, serological identifcation of antigens by
recombinant cDNA expression cloning; SH3BP5, SH3 domain-binding
protein 5; SH3GL1, SH3-domain GRB2- like 1; s-GADD34-Abs, serum
anti-GADD34 antibodies; SNCA, α-synuclein; SOSTDC1, sclerostin
domain-containing protein 1; TACSTD2, tumor-associated calcium
signal transducer 2; TRIM21, tripartite motif-containing 21; TUBB2C,
tubulin beta-2C; ZnT8, zinc transporter 8.
Introduction
Parkinson disease (PD) is a common neurodegenerative disease
with core pathological features including progressive dopaminergic
Abstract
Background: Growth arrest and DNA-damage-inducible gene 34 (GADD34) has been identifed as an antigen
by serological identifcation of antigens by cDNA expression cloning (SEREX) using the sera of patients with
atherosclerosis. It is possible that GADD34 is associated with atherosclerosis-related diseases such as diabetes
mellitus (DM), acute-phase cerebral infarction (aCI), cardiovascular disease (CVD), and chronic kidney disease
(CKD) as well as endoplasmic reticulum stress-related Parkinson disease (PD).
Methods: GADD34 protein was bacterially expressed and purifed. Amplifed luminescent proximity homogeneous
assay (AlphaLISA) was used to evaluate serum antibody levels against GADD34 protein in serum samples.
Results: AlphaLISA revealed signifcantly higher serum antibody levels against GADD34 protein in patients
with DM, aCI and CVD than those in healthy donors (HDs). The difference in levels between DM and HD was more
prominent than that between aCI or CVD and HDs. The anti-GADD34 antibody levels were also elevated in the sera
of diabetic CKD patients; thus, the anti-GADD34 antibodies appeared to be the most associated with DM, which
is also a risk factor of PD. Anti-GADD34 antibody levels were also higher in patients with PD but not in those with
amyotrophic lateral sclerosis as compared with those in HDs.
Conclusion: Anti-GADD34 antibody may be a useful diagnostic tool for DM and PD. GADD34 may account for
the pathophysiological relationship between DM and PD.
Identification of Serum Anti-GADD34 Antibody as a Common Marker of
Diabetes Mellitus and Parkinson Disease
Kazuo Sugimoto
1,2,#
, Go Tomiyoshi
2,3,#
, Masahiro Mori
1
, Satoshi Kuwabara
1
, Shigeki Hirano
1
, Setsu Sawai
1
, Minako Beppu
1,4
, Mayumi Muto
1
,
Akiyuki Uzawa
1
, Kenichiro Kitamura
5
, Minoru Takemoto
6
, Akiko Hattori
6
, Masashi Yamamoto
6
, Kazuki Kobayashi
6
, Harukiyo Kawamura
6
,
Ryoichi Ishibashi
6
, Koutaro Yokote
6
, Seiichiro Mine
7,8,9
, Toshio Machida
9
, Eiichi Kobayashi
7
, Yoichi Yoshida
7
, Tomoo Matsutani
7
, Yasuo
Iwadate
7
, Yoshio Kobayashi
10
, Rika Nakamura
2,3
, Natsuko Shinmen
2,3
, Hideyuki Kuroda
3
, Hao Wang
2,11
, Xiao-Meng Zhang
2
and Takaki Hiwasa
2
*
1
Department of Neurology, Graduate School of Medicine, Chiba University, Chiba 260-8670, Japan
2
Department of Biochemistry and Genetics, Graduate School of Medicine, Chiba University, Chiba 260-8670, Japan
3
Medical Project Division, Research Development Center, Fujikura Kasei Co., Saitama 340-0203, Japan
4
Department of Molecular Diagnosis, Graduate School of Medicine, Chiba University, Chiba 260-8670, Japan
5
Department of Internal Medicine 3, University of Yamanashi School of Medicine, Yamanashi 409-3898, Japan
6
Department of Clinical Cell Biology and Medicine, Graduate School of Medicine, Chiba University, Chiba 260-8670, Japan
7
Department of Neurological Surgery, Graduate School of Medicine, Chiba University, Chiba 260-8670, Japan
8
Department of Neurological Surgery, Chiba Prefectural Sawara Hospital, Chiba 287-0003, Japan
9
Department of Neurological Surgery, Chiba Cerebral and Cardiovascular Center, Chiba 290-0512, Japan
10
Department of Cardiovascular Medicine, Graduate School of Medicine, Chiba University, Chiba 260-8670, Japan
11
Department of Anesthesia, The First Affliated Hospital, Jinan University, Guangzhou 510630, P. R. China
Ϯ
Authors contributed equally