ELSEVIER Mutation Research 334 (1995) 175-183
Environmental Mutagenesis
Cytogenetic study of tuberculosis patients
before and after tuberculostatic drug treatment
Abdullah Ekmek§i *, Ay~e ~ayli
Department of Medical Biology and Genetics, Faculty of Medicine, Gazi University, 06510 Besevler, Ankara, Turkey
Received 21 September 1993; revision received 7 June 1994; accepted 8 August 1994
Abstract
Cytogenetic analyses were carried out in lymphocytes of 15 untreated tuberculosis (tb) patients and 15 other tb
patients who had received combined tuberculostatic chemotherapy HRZ (isoniazid + rifampicin + pyrazinamide) for
2 months. The frequency of chromosomal aberrations and sister-chromatid exchanges (SCEs) did not show any
statistically significant differences in the patients before treatment and after exposure to combined HRZ therapy as
compared to controls (p > 0.05). However, we observed that the mitotic index was significantly decreased in both
groups (p < 0.05). Based on the results of the present study, we believe there is no indication for a chromosome
damaging effect of HRZ and their metabolites in human lymphocytes in vivo after treatment of tuberculosis patients
with optimum doses.
Keywords: Tuberculosis chemotherapy; Cytogenetic effects
1. Introduction
Despite availability of highly effective
chemotherapy, the tubercle bacillus continues to
cause devastating illness in the world with wide
socio-economic implications even a century after
its first identification (WHO, 1983). In the treat-
ment of tuberculosis, drugs are usually prescribed
in certain combinations. It is well known that in
combination drugs may act synergistically, antag-
onistically or additively. Therefore, the combined
effects of different drugs need to be the same as
* Corresponding author. Fax 90 312 213 4338.
their individual genetic effects, and some of these
have been tested by various research groups.
However, there are some contradictory cytoge-
netic findings about the endo/exotoxins released
by Mycobacterium tuberculosis in untreated pa-
tients (Gapol Rao et al., 1990) and some com-
monly used antitubercular drugs individually
(Stott et al., 1976; Bauchinger et al., 1978) or in
combination with others (Bauchinger et al., 1978;
Jaju et al., 1981, 1983; Roman et al., 1983). More-
over, chromosome damage induced by tubercle
bacilli (Gapol Rao et al., 1990) and by antituber-
cular drugs (Gapol Rao et al., 1991) was reported
by the same research group in different times.
This study was designed to compare the chro-
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