Vol. 100, No. 2,1981
May 29, 1981
BIOCHEMICAL AND BIOPHYSICAL RESEARCH COMMUNICATIONS
Pages 769~777
IN VITRO EFFECT OF PROSTAGLANDINS ON CORTICOSTERONE
AND ALDOSTERONE PRODUCTION BY FROG INTERRENAL GLAND.
Catherine Delarue, Isabelle Perroteau, Fran§ois Leboulenger,
Pierre Netchitailo, Philippe Leroux, Sylvie Jggou,
Alain Bglanger~, Marie-Christine Tonon and Hubert Vaudry.
Groupe de Recherche en Endocrinologie Mol~culaire,
ERA CNRS 891, Laboratoire d'Endocrinologie,
Facultg des Sciences, 76130 - Mont-Saint-Aignan, France
and ¶Centre de Recherche en Endocrinologie Mol~culaire, Le
Centre Hospitalier de l'Universit~ Laval, Boulevard Laurier,
Qugbec, GIV 4G2 Canada.
Received April 7,1981
SUMMARY.
In order to elucidate the role of prostaglandins of the E and F series
on adrenal steroidogenesis, we have studied corticosterone and aldosterone
production by frog interrenal. Rana ridibunda interrenal dice were perifused
with amphibian culture medium for ten hours. Corticosterone and aldosterone
concentrations were measured in the effluent perifusate using sensitive and
specific radioirmnunoassay methods. Perifusion of interrenal fragments with
increasing concentrations of PGE 1 and PGE 2 (ranging from 8.8 nM to 2.8 ~M)led
to a dose-related increase in both corticosterone and aldosterone biosynthesis,
the magnitude of the stimulation being 1.3 fold higher for aldosterone than
for corticosterone. High concentrations of PGF2~ (2.8 ~M) were only responsible
for a slight increase in corticosteroid biosynthesis while PGFI~ was almost
inactive. Indomethacin an inhibitor of prostaglandin biosynthesis caused a
marked decrease of spontaneous production of corticosterone (-84%) and aldo-
sterone (-75%) but did not alter the stimulation of steroidogenesis induced
by ACTH. From these data, it was concluded that ]) exogenous prostaglandins
control cortieosteroid production in amphibia ; 2) endogenous prostaglandins
are required for spontaneous biosynthesis of corticosteroids ; 3) endogenous
prostaglandins are not involved in ACTH-induced steroidogenesis.
The studies which have been designated to investigate the role of prosta-
glandins (PGs) in the regulation of adrenocortical steroidogenesis have yield-
ed to conflicting results. According to the type of PGs used, to the animals
studied and to the technique employed, both inhibition (]), non stimulation
(2, 3) and stimulation (4-6) have been reported. Concurrently, controversial
results have been obtained with indomethacin (IDM), a potent inhibitor of PG
synthesis. Gallant and Brownie (2) have shown that, in vivo, IDM does not
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