© Freund Publishing House Ltd., London Journal of Pediatric Endocrinology & Metabolism, 13, 1493-1502 (2000)
Hypothalamic Growth Hormone-Insulin-like Growth Factor-I Axis
across the Human Life Span
Ezio Ghigo, Emanuela Arvat, Laura Gianotti, Fabio Lanfranco, Fabio Broglio, Gianluca Aimaretti,
Mauro Maccario and Franco Camanni
Department of Internal Medicine, Division of Endocrinology, University of Turin, Turin, Italy
ABSTRACT
The activity of the growth hormone (GH)-
insulin-like growth factor-I (IGF-I) axis under-
goes marked variations across the human life
span, mainly reflecting age-related changes in
the neural control of somatotroph function.
IGF-I secretion generally reflects GH status,
except in newborns, who secrete high levels of
GH but low levels of IGF-I. Changes in the
gonadal steroid milieu, particularly estradiol,
play a major role in the enhanced activity of the
GH-IGF-I axis at puberty and probably reflect
further changes in the neuroendocrine control of
somatotroph secretion. The change in respon-
siveness of somatotrophs to various stimuli,
including GHRH, is not as marked as the
spontaneous secretion of GH at puberty. How-
ever, in childhood, somatotrophs are unusually
refractory to the somatostatin-mediated negative
GH autofeedback mechanism. Normal children
show normal responsiveness to the stimulatory
influence of α-adrenergic and cholinergic ago-
nists, galanin and arginine, but the activating
effect of these stimuli on somatotroph secretion
is reduced in elderly individuals, with the
notable exception of arginine. Arginine potenti-
ates both spontaneous and GHRH-induced GH
secretion to the same extent in normally growing
children, adults and elderly individuals, indi-
cating that the releasable pool of GH is generally
preserved across the human life span. Thus, the
reduction in spontaneous and GHRH-induced
Reprint address:
E. Ghigo
Division of Endocrinology
Ospedale Molinette
C.so Dogliotti 14
10126 Torino, Italy
e-mail: ezio.ghigo@unito.it
GH secretion in the elderly probably reflects
age-related changes in neurotransmitter control,
leading to GHRH hypoactivity and absolute or
relative somatostatin hyperactivity in the aged
hypothalamus. Cholinergic impairment in the
aging brain probably involves hypothalamic
pathways and leads to decreased activity of the
GH-IGF-I axis in normal and elderly indivi-
duals, as well as in individuals with premature
brain aging. However, there is evidence indi-
cating that age-related variations in the activity
of the natural GH-secretagogue ligand(s) at the
hypothalamic level could also play a role in the
age-dependent changes in the GH-IGF-I axis.
KEY WORDS
growth hormone, human life span, insulin-like
growth factor-I, neurotransmitters
INTRODUCTION
The activity of the growth hormone (GH)-
insulin-like growth factor-I (IGF-I) axis undergoes
marked variations across the human life span. GH
secretion is transiently elevated at birth, markedly
increases at puberty, then progressively decreases to
very low levels in senescence
1
'
9
. IGF-I secretion
generally reflects the GH status, except in new-
borns, who secrete high levels of GH but low levels
of IGF-I
8
'
10
.
It is well known that the increased activity of the
GH-IGF-I axis at puberty is coupled with gonadal
development and is devoted to sustaining the
pubertal growth spurt
1
'
11,12
. However, it is now
common opinion that a component of the
senescence-related changes in body composition,
the functions of various systems (e.g. the cardio-
vascular system) and metabolism could reflect the
VOLUME 13, SUPPLEMENT 6, 2000 1493
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