Magnesium Research 2012; 25 (2): 89-98 ORIGINAL ARTICLE
Increase in intra-abdominal pressure
raises brain venous pressure, leads to
brain ischaemia and decreases brain
magnesium content
Bozena Jarosz
1
, Wojciech Dabrowski
2
, Andrzej Marciniak
3
, Piotr Wacinski
4
,
Ziemowit Rzecki
2
, Edyta Kotlinska
2
, Jacek Pilat
5
1
Department of Neurosurgery and Paediatric Neurosurgery, Medical University of
Lublin; Poland;
2
Department of Anaesthesiology and Intensive Therapy, Medical Uni-
versity of Lublin, Poland;
3
Department of Physiology, Medical University of Lublin,
Poland;
4
Department of Cardiology, Medical University of Lublin; Poland;
5
Department of
General Surgery, Transplantology and Clinical Nutrition, Medical University of Lublin,
Poland
Correspondance : Wojciech Dabrowski, Department of Anaesthesiology and Intensive Therapy, Medical
University of Lublin; Jaczewskiego 8; 20-954 Lublin; Poland.
<w.dabrowski5@yahoo.com>
Abstract. Background. Intra-abdominal hypertension (IAH) may increase
brain venous pressure, which may lead to brain injury. The aim of the present
study was to analyse the effect of IAH on brain venous pressure and brain total
and ionised magnesium (tMg and iMg), calcium (Ca) and zinc (Zn) contents in
rats. Material and methods. Forty four adult Wistar rats were examined. Ani-
mals were divided into two groups: control, and IAH: rats with intra-abdominal
pressure (IAP) elevated to 25 mmHg. IAP was measured directly in the abdomi-
nal cavity. After retrograde cannulation of the jugular vein, the jugular venous
pressure (JVP) was measured as the brain venous pressure. JVP and IAP were
noted after induction of anaesthesia, immediately following induction of IAH
and 90 min after induction of IAH. In all rats, brains were removed for biochemi-
cal and histological analysis. Results. Biochemical analysis was performed in
30 rats, histological visualisation in 14. IAP elevated to 25 mmHg increased
JVP in the IAH group. After 90 min, JVP decreased; however, its value was still
higher compared with pre-IAH. In the IAH group, tMg and iMg were signifi-
cantly lower than in the control group. Moreover, Ca and Zn levels were higher
in the IAH group compared with the control group. The histological examina-
tion showed changes indicative of ischaemic neuronal cell stress. Conclusions.
Firstly, increase in IAP elevates JVP. Secondly, raised JVP decreases tMg and
iMg. Thirdly, raised JVP increases the Ca and Zn content in the rat brain.
Fourthly, IAH leads to changed characteristics of brain ischaemia.
Key words: intra-abdominal pressure, intra-abdominal hypertension, brain
ischaemia, brain venous hypertension, magnesium, calcium, zinc
Intra-abdominal pressure (IAP) is defined as
the steady-state pressure in the peritoneal cavity
[1]. The normal value of IAP ranges from
5 to 7 mmHg, but its elevation to as high
as 12 mmHg is not clinically important. IAP
higher than 12 mmHg is called intra-abdominal
hypertension (IAH); however, organ dysfunction
resulting from IAH is most frequently observed
at IAP higher than 20 mmHg and is referred
to as abdominal compartment syndrome (ACS)
doi:10.1684/mrh.2012.0310
89
To cite this article: Jarosz B, Dabrowski W, Marciniak A, Wacinski P, Rzecki Z, Kotlinska E, Pilat J. Increase in intra-abdominal
pressure raises brain venous pressure, leads to brain ischaemia and decreases brain magnesium content. Magnes Res 2012; 25(2):
89-98 doi:10.1684/mrh.2012.0310
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