[CANCER RESEARCH 37, 3578-3584, October 1977]
SUMMARY
Regulation ‘of the growth and production of granubocyte
colony-stimulating activity (CSA) by WEHI-3 cells, a lyso
zyme-secreting mouse cell line adapted to culture, was
investigated in vitro. WEHI-3 cloning efficiency is not en
hanced by exogenously added CSA. However, WEHI-3 don
ing efficiency in agar was suppressed by an activity in
human polymorphonucleam neutmophil extract (colony-in
hibiting activity) which inhibits endogenous WEHI-3 CSA
production. The addition of increasing concentrations of
WEHI-3- or L cell-conditioned medium containing CSA to
CIA-depressed WEHI-3 agar cultures resulted in graded in
creases of cloning efficiency to that of the untreated sam
pIe. Testosterone, Deca-Dumabolin, and bacterial lipopoly
sacchanide increased production of CSA by WEHI-3 cells
and overcame colony-inhibiting activity-mediated suppres
sion of CSA production, even when activating agents were
added 1 day after the addition of colony-inhibiting activity.
The activating agents had no direct stimulatomy effect on
normal mouse marrow colony-forming cells and did not
enhance CSA activity. WEHI-3 cells respond to growth in
hibitory and stimulatory activities and can serve as an in
vitro model for studying the regulation of neoplastic cells.
INTRODUCTION
Human and mouse bone marrow contain CFU-c3 capable
of in vitro colony formation of gmanubocytesand/or mono
cytes in agam. CFU-c are believed to be granubocytic and
monocytic stem cells (14). CFU-c from normal donors and
patients with acute and chronic leukemia depend on a
family of macromolecules termed CSA (15) for growth and
differentiation in vitro. In addition, CSA production by CSA
producing cells from normal and leukemic patients can be
suppressed by colony-inhibiting activity derived from PMN
(1, 2).
The availability of continuously propagated cell lines,
manifesting growth and regulatory responses similar to hu
man leukemic stem cells and CSA-producing cells, would
be extremely helpful for undemstanding the mechanisms
involved in the regulation of neoplastic cells. WEHI-3 cells,
I This work was supported in part by National Cancer Institute Grants CA
08748, CA-17353, and CA-17085, National Science Foundation Grant BMS
75-19734, and the Gar Reichman Foundation.
2 Special Fellow of the Leukemia Society of America. To whom requests
for reprints should be addressed.
3 The abbreviations used are: CFU-c, colony-forming cells; CSA, colony
stimulating activity; PMN, polymorphonuclear neutrophils; FCS, fetal calf
serum; CML, chronic myelogenous leukemia; LPS, Iipopolysaccharide.
Received April 11, 1977; accepted July 7, 1977.
originally obtained as a myebomonocytic leukemia from
BALB/c mice (28) and now adapted to culture (19), were
studied for their responsiveness to substances that influ
ence normal and leukemic human cells.
The original population of WEHI-3 cells contained neo
plastic granulocytic-monocytic stem cells and cells capable
of elaborating CSA (13, 16). WEHI-3 stem cells could prolif
ematewithout added CSA but its cloning efficiency could be
enhanced by exogenous CSA. The WEHI-3 cell line then
appeared to lose all responsiveness to exogenous CSA (11,
14), although production of CSA was maintained (19).
In this report we describe that WEHI-3 cells can still me
spond to exogenous CSA, but endogenous CSA production
must first be suppressed by colony-inhibiting activity. In
addition, WEHI-3 cells represent a faithful model for CSA
producing cells. They respond, as do normal human cells,
with increased CSA production after activation by testostem
one, Deca-Dumabolin, and bacterial LPS.
MATERIALSAND METHODS
Cells Cultured
Cell Lines. WEHI-3 cells were originally obtained from
BALB/c mice after induction of myebomonocytic leukemia
with mineral oil (28). The cell line has been growing in
culture since January 1974. WEHI-3 cells are phagocytic,
contain Fc receptors, and synthesize lysozyme and CSA (19,
20). Monocyte PU5-1.8 and T lymphocyte EL4 tumor cell
lines have been described by us previously (19, 20). Macro
phage tumor line RAW264 was induced by Abelson leuke
mia virus.4 Rauschen lymphoma RBL3 and chemically in
duced L1210 lymphoma were obtained from Dr. K. Chang,
National Cancer Institute, Bethesda, Md. All cell lines grow
in culture with doubling times of 12 to 24 hr. The cells often
attach firmly to tissue culture dishes, particularly upon dilu
tion at low cell densities or in the absence of serum. More
common is a mixture of attached cells and loosely adherent
or floating, round cells, with the latter predominating at
high cell densities. Both the adherent and nonadherent
cells can be used to propagate the lines, and no difference
in effectom functions has been noted between the adherent
and nonadherent forms (21). The cell lines, that have been
adapted to culture, form solid tumors and ascites in mice
ofthe properstmain (21).
Normal Cells. CFU-c were obtained from the bone mar
mowof C57BL/6mice. Bonemarrowcells suspendedin
4 P. Ralphand I. Nakoinz,manuscriptin preparation.
3578 CANCERRESEARCHVOL. 37
In Vitro Regulation of a Mouse Myelomonocytic Leukemia Line
Adapted to Culture1
Hal E. Broxmeyer2 and Peter Ralph
MemorialSloan-KetteringInstitutefor CancerResearch,NewYork,New York10021
Research.
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