Current Radiopharmaceuticals
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Current Radiopharmaceuticals, 2021, 14, 145-153
145
RESEARCH ARTICLE
Calcium Carbonate Microparticles as Carriers of
224
Ra: Impact of Specific
Activity in Mice with Intraperitoneal Ovarian Cancer
Ruth Gong Li
1,2,3
, Elisa Napoli
1,2,3
, Ida Sofie Jorstad
1
, Tina Bjørnlund Bønsdorff
1
, Asta Juzeniene
3
,
Øyvind Sverre Bruland
2,4
, Roy Hartvig Larsen
1
and Sara Westrøm
1,*
1
Oncoinvent AS, Oslo, Norway;
2
Institute of Clinical Medicine, University of Oslo, Oslo, Norway;
3
Department of Radi-
ation Biology, Institute of Cancer Research, the Norwegian Radium Hospital, Oslo University Hospital, Oslo, Norway;
4
Department of Oncology, the Norwegian Radium Hospital, Oslo University Hospital, Oslo, Norway
ARTICLE HISTORY
Received: May 29, 2020
Revised: September 28, 2020
Accepted: October 06, 2020
DOI:
10.2174/1874471013666201201102056
Abstract: Background: Patients with advanced-stage ovarian cancer face a poor prognosis be-
cause of recurrent peritoneal cavity metastases following surgery and chemotherapy. Alpha-emit-
ters may enable the efficient treatment of such disseminated diseases because of their short range
and highly energetic radiation. Radium-224 is a candidate α-emitter due to its convenient 3.6-day
half-life, with more than 90% of the decay energy originating from α-particles. However, its inher-
ent skeletal accumulation must be overcome to facilitate intraperitoneal delivery of the radiation
dose. Therefore,
224
Ra-labeled CaCO
3
microparticles have been developed.
Objective: The antitumor effect of CaCO
3
microparticles as a carrier for
224
Ra was investigated,
with an emphasis on the ratio of activity to mass dose of CaCO
3
, that is, specific activity.
Methods: Nude athymic mice were inoculated intraperitoneally with human ovarian cancer cells
(ES-2) and treated with a single intraperitoneal injection of
224
Ra-labeled CaCO
3
microparticles
with varying combinations of mass and activity dose, or cationic
224
Ra in solution. Survival and
ascites volume at sacrifice were evaluated.
Results: Significant therapeutic effect was achieved for all tested specific activities ranging from
0.4 to 4.6 kBq/mg. Although treatment with a mean activity dose of 1305 kBq/kg of cationic
224
Ra
prolonged the survival compared with the control, equivalent median survival could be achieved
with
224
Ra-labeled microparticles with a mean dose of only 420 kBq/kg. The best outcome was
achieved with the highest specific activities (2.6 and 4.6 kBq/mg).
Conclusion: Radium-224-labeled CaCO
3
microparticles present a promising therapy against can-
cer dissemination in body cavities.
Keywords: Alpha therapy, calcium carbonate, microparticles, radium-224, 224Ra, peritoneal carcinomatosis, intraperitoneal,
ovarian cancer.
1. INTRODUCTION
Ovarian cancer presents numerous challenges, the most
pressing of those being the survival rate; the worldwide five-
year net survival is in the range of 30–50% [1]. Most cases
are diagnosed at an advanced stage, with the disease dissemi-
nated within the peritoneal cavity [2], which correlates to a
poor prognosis [3]. The standard treatment for advanced-s-
tage disease is cytoreductive surgery in combination with
platinum-based chemotherapy [4]. Although this treatment
regime initially gives some benefits, only a minority of wom-
en with advanced ovarian cancer survive beyond five years
after diagnosis [5], emphasizing the need for innovative ther-
apies.
*Address correspondence to this author at Oncoinvent AS, Oslo, Norway;
Tel: +47 913 97 563; E-mail: westrom@oncoinvent.com
Patients who have undergone surgical resection of all vis-
ible tumors are at high risk of subsequent peritoneal relapse
related to the presence of residual microscopic tumor de-
posits and free-floating cancer cells [6]. Intraperitoneal (IP)
treatment with various radionuclides has been evaluated clin-
ically for the treatment of disseminated ovarian cancer
[7-12]. Historically, radiocolloids based on β-emitters
198
Au
and
32
P have been used with some success [13] but were
eventually abandoned and replaced with chemotherapy be-
cause of the more frequent incidence of late adverse effects
[7, 14]. The use of colloids was subsequently exchanged
with various monoclonal antibodies to which β-emitters
were coupled, and several phase II clinical trials have been
performed [8-11]. Unfortunately, the only phase III clinical
trial investigating
90
Y coupled to an anti-MUC1 antibody
failed to confirm both improved survival and reduced time
to relapse [12].
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