Pediatric Diabetes 2011: 12: 536 – 546
doi: 10.1111/j.1399-5448.2010.00739.x
All rights reserved
© 2011 John Wiley & Sons A/S
Pediatric Diabetes
Original Article
Parathormone – 25(OH)-vitamin D axis
and bone status in children and adolescents
with type 1 diabetes mellitus
Hamed EA, Abu Faddan NH, Adb Elhafeez HA, Sayed D.
Parathormone – 25(OH)-vitamin D axis and bone status in children and
adolescents with type 1 diabetes mellitus.
Pediatric Diabetes 2011: 12: 536 – 546.
Background: Skeletal involvement in patients with type 1 diabetes mellitus
(T1DM) has complex pathogenesis and despite numerous researches on this
problem, many questions remain unanswered.
Objective: This study aimed to assess bone status by measurement
parathormone (PTH), 25-hydroxy vitamin D [25(OH)D] serum levels in
children and adolescents with T1DM and its relation to insulin-like growth
factor-1 (IGF-1), disease duration, puberty stage, and metabolic control.
Patients and methods: This study included 36 children and adolescents with
T1DM and 15 apparently healthy controls. Serum levels of 25(OH)D, PTH,
IGF-1 measured using enzyme-linked immunosorbent assay (ELISA), while
glycosylated hemoglobin (HbA1c), calcium (Ca), inorganic phosphorus (PO
4
)
using autoanalyzer. Bone quality assessed using dual energy X-ray
absorptiometry (DEXA).
Results: Diabetic patients showed significant increase in PO
4
and PTH levels,
while significant decrease in Ca, IGF-1, and 25(OH)D serum levels. As much
as 52.8% of patients showed reduced 25(OH)D, and 30.65% showed elevated
PTH serum levels. In diabetic patients, abnormal bone status
(osteopenia-osteoporosis) found mostly in total body (94.40%) then
lumber-spine (88.90%), ribs (88.90%), pelvis (86.10%), thoracic-spine
(80.60%), arms (80.60%) and legs (77.80%), while head bones showed no
abnormalities. Long diabetic duration had negative; meanwhile PTH, onset
age, and puberty age had positive impact on bone status.
Conclusions: Children and adolescent with T1DM have abnormal bone
status mostly in axial skeleton which may be contributed to impairment of
formation of 25(OH)D and IGF-1. Physical activity, calcium and vitamin D
supplement seem important in T1DM. Elevated serum PTH level in diabetic
patients is not uncommon and its positive correlation with bone status needs
further investigations.
Enas A Hamed
a
, Nagla H
Abu Faddan
b
, Hebh A Adb
Elhafeez
c
and Douaa Sayed
d
a
Department of Physiology, Faculty of
Medicine, Assiut University, Assiut,
P. O. Box 71526, Egypt;
b
Department
of Pediatrics, Pediatric Assiut
University Hospital, Assiut, P. O. Box
71526, Egypt;
c
Department of Clinical
Pathology, Assiut University Hospital,
Assiut, P. O. Box 71526, Egypt; and
d
Department of Clinical Pathology,
South Egypt Cancer Institute, Assiut
University, Assiut, P. O. Box
71526, Egypt
Key words: bone status – 25(OH)D –
IGF-1 – PTH – TIDM
Corresponding author:
Assist. Prof. Enas A Hamed, MD,
Department of Physiology,
Faculty of Medicine,
Assiut University,
Assiut, P. O. Box 71526, Egypt.
Tel: +2 0164743592;
fax: +2 088 2333327;
e-mail: eah3a2010@yahoo.com
Submitted 22 July 2010.
Accepted for publication 6 October
2010
The prevalence of type 1 diabetes mellitus (T1DM)
in childhood is increasing with a worldwide annual
increase estimated at 3% (range 2–5%) (1). T1DM
has negative effects on bone health and leads to an
increase in fracture risk among middle aged and older
individuals (2). However, there is still some debate
about the effect of diabetes on bone status during
childhood and adolescence (3, 4). Moreover, there is
no general agreement on the relative importance of
several diabetes – specific characteristics, such as age of
onset, diabetic duration, glycemic control, and insulin
regimen on bone health (5). Puberty has a key role
in bone development. Skeletal mass approximately
doubles at the end of adolescence (6). The pubertal
phase is characteristically associated with a reduction
in insulin sensitivity, which is known to be more severe
in patients with T1DM, and might negatively influence
growth and height gain (7). Identifying risk factors
that predispose to a low bone mineral density (BMD)
in diabetic patients is therefore desirable.
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