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Analysis of DNA Restriction Fragment Length Polymorphism Extends the
Evidence for Breast Milk Transmission in Streptococcus agalactiae Late-Onset
Neonatal Infection
Edouard Bingen, Erick Denamur,
Nicole Lambert-Zechovsky, Yannick Aujard,
N alma Brahimi, Pierre Geslin, and Jacques Elion
Laboratoires de Bacteriologic and Biochimie Genhique. Service de
Neonatologie et I NSERM U J20. Hiipital Robert Debre, Paris;
Laboratoire de Bacteriologic. Centre Hospitalier
Intercommunal de Creteil, France
Analysis of restriction fragment length polymorphism (RFLP) of total DNA and of ribosomal
DNA (ribotyping) was used to document four cases of Streptococcus agalactiae mother-to-infant
transmission potentially associated with ingestion of infected mother's milk. Twenty strains were
analyzed. Ten strains were mother-baby pairs, five from the milk of five mothers, four from their
neonates with late-onset infection, and one from a colonized neonate. All mothers had early
postpartum mastitis. Ten unrelated strains were studied for comparison. In each case, the two
strains of each mother-baby pair produced identical RFLP patterns of total DNA. The 10 unre-
lated strains generated 10 different patterns, one of which, though, was observed in one of the
mother-baby pairs. Ribotyping was less discriminative than total DNA RFLP analysis (6 differ-
ent patterns vs. 13). These data extend the evidence for breast milk transmission in S. agalactiae
late-onset neonatal infection.
Streptococcus agalactiae is the major cause of severe infec-
tion during the first 2 months of life [I]. Acquisition of S.
agalactiae by neonates in the early-onset type of infection is
clearly vertical from the maternal genital tract in utero or
during passage through the birth canal [I]. But the mode of
transmission of S. agalactiae in infants with late-onset dis-
ease is not so well defined. Previous studies have provided
evidence that infants acquire the organism from their
mothers [1], nosocomially, via the contaminated hands of
nursery personnel [2], or, occasionally, from community
contacts [3]. It has been also suggested that maternal strepto-
cocci may be transmitted by breast-feeding [4-6]. Study of
the modes of transmission in these infections has been some-
what hampered by the lack of a sufficiently discriminatory
typing system.
Received 3 July 1991; revised 23 September 1991.
Reprints or correspondence: Dr. E. Bingen. Laboratoire de Bacteriologic,
Hopital Robert Debre, 48 Bd Serurier, 75019 Paris, France.
The Journal of Infectious Diseases 1992;165:569-73
© 1992 by The University of Chicago. All rights reserved.
0022-1899/92/6503-0027$01.00
In this study, we analyzed the restriction fragment length
polymorphism (RFLP) of total DNA and of ribosomal DNA
(rDNA) regions (ribotyping) to document four cases of S.
agalactiae vertical mother-to-infant transmission associated
with ingestion of infected mother's milk.
Materials and Methods
Patient population. Patients were from the obstetric and
newborn services and from the neonatal intensive care unit at
the Hopital Robert Debre, Paris, a children's hospital. Five
mother-baby pairs were studied (table 1). All mothers had post-
partum mastitis with erythema, pain, and tenderness in the sub-
areolar region but all continued breast-feeding until infant col-
onization or infection was ascertained. Two patients subse-
quently developed a breast abscess. Of the five neonates, four
had a late-onset infection with meningitis (one case), arthritis
(one case), and urinary tract infection (two cases). One neonate
was colonized with S. agalactiae. The mean age of the four neo-
nates at the onset of infection was 11 days (range, 5-19).
Bacterial strains. Twenty strains of S. agalactiae were se-
lected for study. Ten were mother-baby pairs (table 1). Five
were from maternal breast milk of five mothers with a mean
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