~ Pergamon
0043-1354(95)00189-1
Wat. Res. Vol. 30, No. 2, pp. 481--485, 1996
Copyright © 1996 ElsevierScienceLtd
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RESEARCH NOTE
BIODEGRADATION OF MICROCYSTIN-LR BY
INDIGENOUS MIXED BACTERIAL POPULATIONS
I. T. COUSINS*, D. J. BEAL1NG, H. A. JAMESf and A. SUTTON
WRc plc, Henley Road, Medmenham, Marlow, Bucks SL7 2HD, England
(First received July 1994; accepted in revised form August 1995)
Abstract--Microcystin-LR is a potent mammalian toxin which is known to have been responsible for the
deaths of domesticated animals, and consequently there is concern as to its environmental fate. In
laboratory experiments conducted using low levels (10 #g I-~) of microcystin-LR in reservoir water,
primary biodegradation of the toxin was shown to occur in less than 1 week. An experiment designed
to ascertain the degree of mineralisation of microcystin-LR, using natural microbial populations under
aerobic conditions, showed that the peptide ring appears to be fairly resistant to biodegradation, as the
degree of mineralisation was not high. However, as the toxicity of microcystin-LR is critically dependent
on the orientation of the peptide ring with respect to the "Adda" (the fl-amino acid which is unique to
the blue-green algal toxins) side-chain, and it appears that this side chain is affected during biodegradation,
it seems likely that a reduction in toxicity occurs.
Key words--blue-green algae, algal toxins, microcystin-LR, biodegradation
INTRODUCTION
Microcystin-LR is a toxic cyclic heptapeptide which
may be produced by some strains of various
blue-green algae e.g. Microcystis, Oscillatoria and
Anabaena species. Low levels of the toxin may be
released into water bodies during the normal growth
and turnover of blue-green algal populations and
high levels may result during algal blooms. Deaths of
domestic animals, following the ingestion of toxic
blue-green algal scums from algal blooms, have
demonstrated that under certain circumstances very
high concentrations of the toxin are produced and
can be lethal. Information regarding the persistence
of this toxin is necessary to allow decisions to be
taken on the use of water bodies for recreational
purposes following blue-green algal blooms. Such
information is also necessary to allow decisions to be
taken on the requirements for water treatment.
Although microcystin-LR is the most commonly
found microcystin (Carmichael, 1992), many other
microcystins are known, and blue-green algae which
produce hepatotoxins usually produce a mixture of
different microcystins. However, as they are all cyclic
heptapeptides containing an unusual B-amino acid
(3 - amino - 9 - methoxy - 10 - phenyl - 2,3,8 - trimethyl -
deca-4,6-dienoic acid) (Adda), it seems likely that
their degradative behaviour would be similar. Recent
work (Jones and Orr, 1994) showed that Microcystis
aeruginosa persisted for 9 d in a recreational lake
before degradation commenced. After this initial lag
phase, however, degradation was rapid with 90-95%
in 3d.
This paper presents the results from experiments
designed to study the conditions under which
microbial degradation of microcystin-LR occurs. A
recent study (Block et al., 1992) showed that
indigenous mixed bacterial populations (of sus-
pended or attached bacteria) were suitable for
biodegradative studies so water and sediment samples
were obtained from a reservoir which has experienced
algal blooms in the past, for use in laboratory
degradation experiments.
The first of these experiments consisted of an
analytical study to assess the rate of primary
biodegradation of microcystin-LR in reservoir water.
This was followed by an "ultimate" biodegradation
study to establish the extent of mineralisation which
occurred, as a high degree of mineralisation would
indicate that the degradation products would be of
little concern.
*Present address: Institute of Environmental and Biological
Sciences, Lancaster University, Lancaster LAI 4YQ,
England.
tAuthor to whom all correspondence should be addressed.
METHODS
Analytical
Analysis for microcystin-LR was carried out using a
published analytical method (James and James, 1991).
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