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Chapter 8
The Use of Lentiviral Vectors to Obtain Transgenic Rats
Séverine Remy, Tuan Huy Nguyen, Séverine Ménoret,
Laurent Tesson, Claire Usal, and Ignacio Anegon
Abstract
Lentiviral vectors are now well recognized as good vehicles for gene delivery. This is because they can
efficiently transduce both dividing and post-mitotic cells, and stably integrate into the host genome
allowing for long-term expression of the transgene. Their potential utility for the generation of transgenic
animals has been recognized as an attractive and promising alternative to the conventional DNA-
microinjection method which lacks efficiency. The initial success of lentiviral transgenesis in mice
considerably broadened its use in other species, in which classical transgenic techniques are difficult, such
as in the rat.
In this chapter, we describe detailed procedures for both the production of human immunodeficiency
virus-1 (HIV-1)-derived lentiviral vectors and for the generation of transgenic rats by injection of these
vectors into the perivitelline space of fertilized one-cell eggs.
Key words: Lentiviral vectors, Transgenic rat, Lentiviral transgenesis
The growing interest in developing new tools and approaches to
modify the genome of animals has led to the emergence of trans-
genic technologies. The first transgenic mice were generated in
1976 by Jaenish, after the infection of mouse embryos with
Moloney leukemia retroviruses (1). Despite the successful integra-
tion and germ line transmission of exogenous DNA of retroviral
origin, the absence of transgene expression in the founder animals
due to retroviral DNA methylation considerably hampered the use
of oncoretroviruses in animal transgenesis. Subsequently, alterna-
tive methods were developed to generate transgenic animals,
with particular focus on the pronuclear microinjection of DNA.
Since its development by Gordon et al. in 1980 (2), this is the
1. Introduction
I. Anegon (ed.), Rat Genomics: Methods and Protocols, Methods in Molecular Biology, vol. 597
DOI 10.1007/978-1-60327-389-3_8, © Humana Press, a part of Springer Science+Business Media, LLC 2010