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2006 THE AUTHORS
JOURNAL COMPILATION
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2 0 0 6 B J U I N T E R N A T I O N A L | 9 9 , 3 11 – 3 1 4 | doi:10.1111/j.1464-410X.2006.06559.x 311
Original Article
RESULTS AFTER SURGERY FOR CLINICAL T3a PROSTATE CANCER WITH OR WITH NO NEOADJUVANT ADT
HSU
et al.
Comparing results after surgery in patients with clinical
unilateral T3a prostate cancer treated with or without
neoadjuvant androgen-deprivation therapy
Chao-Yu Hsu, Steven Joniau, Tania Roskams*, Raymond Oyen† and Hein Van Poppel
Departments of Urology, *Histopathology and †Radiology, University Hospitals KULeuven, Leuven, Belgium
Accepted for publication 16 August 2006
patients were not treated with nADT and 35
were. The Kaplan–Meier method was used to
calculate survival rates.
RESULTS
With no nADT the biochemical progression-
free survival (PFS) was 59.5%, the clinical PFS
was 95.9%, the cancer-specific survival (CSS)
was 98.7%, and overall survival was 95.9% at
5 years. With nADT, the biochemical PFS was
43.4%, clinical PFS was 77.6%, CSS was
88.7%, and overall survival was 79.8% at
5 years. The positive surgical margin rate with
no nADT and with nADT was 33.5% and
57.1%, respectively, and the respective mean
cancer volume was 6.6 mL and 4.0 mL.
CONCLUSION
nADT can decrease tumour size but does not
reduce the positive surgical margin rate, nor
improve the survival rate in unilateral cT3a
disease. Because of side-effects and
treatment costs, we do not advise nADT in
patients with unilateral cT3a prostate cancer.
KEYWORDS
prostate cancer, cT3, neoadjuvant, androgen-
deprivation therapy
OBJECTIVE
To compare the results in patients with
unilateral cT3 prostate cancer treated with or
with no neoadjuvant androgen-deprivation
therapy (nADT), as nADT might have benefit in
cT2 prostate cancer, but for cT3 tumours its
use remains controversial, and it is unclear
whether it can prevent or delay progression
after surgery.
PATIENTS AND METHODS
Between 1987 and 2004, 235 patients were
assessed as having unilateral cT3 disease by a
digital rectal examination; before surgery, 200
INTRODUCTION
Locally advanced prostate cancer is defined as
cancer that has extended clinically beyond
the prostatic capsule, with invasion of the
pericapsular tissue, apex, bladder neck, or
seminal vesicle, but with no lymph node
involvement or distant metastases. The
treatment of T3 prostate cancer can be radical
prostatectomy (RP), radiotherapy, androgen-
deprivation therapy (ADT), or combinations of
these. Traditionally, T3 prostate cancers are
treated by radiotherapy. Recently, after the
publication of the landmark trials by the
Radiation Therapy Oncology Group and the
European Organisation for the Research and
Treatment of Cancer, the treatment of cT3
prostate cancer has changed towards a
combination of radiotherapy and ADT.
According to the guidelines of the European
Association of Urology, RP is an accepted
option in selected patients with small T3a
tumours, a Gleason score of 5–7, a low PSA
level and a life-expectancy of > 10 years [1].
Surgery for locally advanced prostate cancer
is acceptable. The cancer-specific survival
(CSS) at 5 and 10 years was 85% - 100% and
57% - 100%, respectively, and the respective
overall survival (OS) was > 75% and 60% [2].
From our previous study [3], neoadjuvant ADT
(nADT) can decrease the positive surgical
margin (PSM) rate in cT2 tumours, but this
was not the case in cT3 tumours. nADT might
be of benefit in cT2 prostate cancer, but for
cT3 tumours it remains controversial. ADT can
cause gynaecomastia, hot flashes, and
decrease libido and potency. The main
question is whether the use of nADT can
prevent or delay progression after surgery;
thus, the objective of the present study was to
compare the results in patients with unilateral
cT3 prostate cancer treated with or with
no nADT.
PATIENTS AND METHODS
Between 1987 and 2004, 2273 patients had
RP at our institution; 235 (10.3%) were
assessed as having unilateral cT3 disease by a
DRE, and they were selected for surgery on
the basis of limited, unilateral cT3a, any
Gleason score, any PSA level and an Eastern
Cooperative Oncology Group performance
status of 0–1. Before surgery, 200 patients
(8.8%) were not treated with nADT and 35
were. All patients had RP and bilateral pelvic
lymphadenectomy. The mean (range) follow-
up was 74.7 (7–184) months. The data were
retrieved from patient files and from GPs by
telephone for updated clinical and
biochemical information.
The DRE was performed by one of two senior
urologists. Patients with unilateral T3a
assessed by TRUS but not by DRE were
excluded from the study. No patient received
radiation therapy before surgery, while all
patients had no evidence of nodal disease
or distant metastasis on both contrast-
enhanced CT of the pelvis, and a bone scan.
If necessary, we used additional imaging of
suspect lesions.
Most of the patients were referred from a
local hospital, and different nADT regimens
were used before the patients arrived at our
clinic; the interval from nADT to RP was
6–12 weeks. The last PSA value obtained
before prostatic biopsies was used in the
analysis.