Case Report Fahr Syndrome Unknown Complication: Overactive Bladder Devrim Tuglu, 1 Ercan Yuvanç, 1 Fatih Bal, 1 Yakup Türkel, 2 Ersel DaL, 2 Erdal YJlmaz, 1 and Ertan Batislam 1 1 Department of Urology, Faculty of Medicine, University of Kirikkale, Yahsihan, 71100 Kirikkale, Turkey 2 Department of Neurology, Faculty of Medicine, University of Kirikkale, Turkey Correspondence should be addressed to Devrim Tuglu; merdev2002@yahoo.com Received 7 April 2014; Accepted 8 July 2014; Published 16 July 2014 Academic Editor: Bharat Rekhi Copyright © 2014 Devrim Tuglu et al. his is an open access article distributed under the Creative Commons Attribution License, which permits unrestricted use, distribution, and reproduction in any medium, provided the original work is properly cited. A 38-year-old male patient was admitted to our outpatient department because of frequency and urgency incontinence. During evaluation it was detected that the patient was sufering from frequency which was progressive for one year, feeling of incontinence, and urgency incontinence. here was no urologic pathology detected in patient’s medical and family history. Neurologic consultation was requested due to his history of boredom, reluctance to do business, balance disorders, and recession for about 3 years. Brain computerized tomography (CT) scan revealed that amorphous calciications were detected in the bilaterally centrum semiovale, basal ganglia, capsula interna, thalami, mesencephalon, pons and bulbus, and the bilateral cerebellar hemispheres. We have detected spontaneous neurogenic detrusor overactivity without sphincter dyssynergia ater evaluating the voiding diary, cystometry, and pressure low study. We consider the detrusor overactivity which occurred one year ater the start of the neurological symptoms as the suprapontine inhibition and damage in the axonal pathways in the Fahr syndrome. 1. Introduction Fahr’s syndrome, also referred to as idiopathic basal ganglia calciication (IBGC) or bilateral striopallidodentate calci- nosis, is a disease characterized by symmetric, nonatheroscle- rotic, bilateral vascular calciication of the basal ganglia [1]. Clinical symptoms of IBGC are found in the literature as case reports, because the disease is very rare. Clinically, the most common presentation of IBGC is considered to be Parkinsonism or other hyperkinetic movement disorders (chorea, tremor, dystonia, athetosis, orofacial dyskinesia). he second most common presentation of IBGC is cognitive impairment followed by cerebellar impairment and speech disorder. Psychiatric features, gait disorders, sensory changes, and pain are also reported [2]. In our case, we purpose to report “neurogenic detrusor overactivity,” which is an unknown symptom of Fahr’s syn- drome. 2. Case Report A 38-year-old male patient with a history of boredom, reluctance to do business, balance disorders, and recession for about 3 years was admitted to our outpatient department because of frequency and urgency incontinence. During evaluation it was detected that the patient was sufering from frequency which was progressive since one year, feeling of incontinence and urgency incontinence. here was no urologic pathology detected in patient’s medical and family history. Urine analysis, urine culture, blood urea nitrogen, and creatinine levels revealed normal values. A kidney, ureter, and bladder (KUB) X-ray study is nonpatho- logical. Urinary ultrasound revealed that bilaterally kid- neys and urinary bladder had normal values. Urolowmetry result revealed that volume voided was 224 mL, Qmax was 22 mL/s, Qave was 9 mL/s, and postvoiding volume was 15 mL. We have detected spontaneous neurogenic detrusor overactivity without sphincter dyssynergia ater evaluating the voiding diary, cystometry, and pressure low study. Also neurologic consultation was requested due to his history of boredom, reluctance to do business, balance disorders, and recession for about 3 years. In the neurologic evalu- ation, the patient was well oriented and cooperative. He was appearing apathetic. he minimental test score was 29. Hindawi Publishing Corporation Case Reports in Urology Volume 2014, Article ID 939268, 2 pages http://dx.doi.org/10.1155/2014/939268