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Endocrine, Metabolic & Immune Disorders - Drug Targets, 2015, 15, 000-000 1
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Mediterranean Diet and Low-grade Subclinical Inflammation:
The Moli-sani Study
Marialaura Bonaccio
*
, Chiara Cerletti, Licia Iacoviello and Giovanni de Gaetano
Department of Epidemiology and Prevention, IRCCS Istituto Neurologico Mediterraneo NEUROMED, Pozzilli (IS),
Italy
Abstract: Low-grade chronic inflammation is an underlying pathophysiological mechanism linking risk factors and/or
metabolic disorders to increased risk of chronic degenerative disease. A meat-based pattern, as the Western type diet, is
positively linked to higher levels of some important biomarkers of inflammation, such as C-reactive protein (CRP),
interleukin-6 and fibrinogen. Conversely, a Mediterranean-like eating behavior is associated with lower degree of these
biomarkers thus suggesting an anti-inflammatory action of its main food components. This chapter goes through the most
important investigations addressing the relationship between dietary habits and subclinical inflammation. Attention was
focussed on the findings from the Moli-sani study: this is a large prospective cohort study that recruited 24,325 men
and women from the general population of the Molise Region, a Southern Italian area, with the aim of investigating
genetic and environmental risk/protection factors for cardiovascular and tumor disease. For the first time, the Moli-sani
study carefully investigated the Mediterranean diet as an environmental determinant of both platelet and white blood cell
counts, starting from the hypothesis that a diet rich in healthy compounds could favorably influence the production and/or
the clearance of these two cellular biomarkers of low-grade inflammation. Additionally, evidence from this large Italian
cohort showed that a Mediterranean-like diet was closely associated with relatively lower values of glucose, lipids, CRP,
blood pressure and 10-year cardiovascular risk, while the consumption of healthy foods with high rather than low content in
antioxidant vitamins and phytochemicals was associated with lower blood pressure and CRP plasma levels at least in men.
Keywords: Antioxidants, biomarkers of inflammation, cardiovascular disease, cancer, low-grade inflammation, mediterranean diet.
BIOMARKERS OF CHRONIC LOW-GRADE
INFLAMMATION
Low-grade inflammation is a condition not yet
uniformely defined: several biomarkers and different levels
of their increase have been reported to be associated with a
condition of low-grade inflammation [1]. Here a low-grade
inflammation is intended as a subclinical condition (systemic
or local, often chronic) characterized by increased levels of
plasmatic and/or cellular biomarkers of inflammation (e.g.
C-reactive protein, platelet or leukocyte counts) without any
apparent clinical sign. In the absence of an acute
inflammatory condition (e.g. CRP <10 mg/L or WBC <=10
x10
9
/L), a low-grade inflammation may reflect different
degrees of variance within the normal range of each specific
biomarker.
A low-grade inflammation has also been defined as an
underlying pathophysiological mechanism linking risk
factors or metabolic disorders (as oxidative stress, obesity,
diabetes, dyslipidemia, etc.) to an increased risk of chronic
degenerative disease [1]. C-reactive protein is the best-
studied marker of subclinical inflammation [2] but several
other inflammatory biomarkers have been associated with
*Address correspondence to this author at the Department of Epidemiology
and Prevention, IRCCS Istituto Neurologico Mediterraneo NEUROMED,
Pozzilli (IS), Italy, Via dell'Elettronica, 86077 POZZILLI (IS), Italy;
Tel: +39.0865.929.653; Fax: +39.0865.927.575;
E-mail: marialaura.bonaccio@moli-sani.org
low-grade inflammation. Increased levels of interleukins,
fibrinogen and the adhesion molecules E-selectin, intercellular
adhesion molecule-1 (ICAM-1), and vascular adhesion
protein-1 (VCAM-1), have been shown to be directly
associated and to predict type-2 diabetes, cardiovascular
disease and cancer; conversely, levels of anti-inflammatory
biomarkers, such as adiponectin, have been associated with
the reduction of cardiovascular disease, type-2 diabetes, and
obesity-related cancer [1].
A fatty meal may represent an acute in vivo challenge
that induces a low-grade inflammatory reaction. In a group
of subjects at different degree of cardiovascular risk, the
post-prandial condition, characterized by acutely elevated
levels of triglyceride-rich lipoproteins, was accompanied by
significant changes in haematological parameters: increased
white blood cell counts, in particular granulocytes, increased
platelet count and decreased mean platelet volume [3, 4].
The changes were statistically significant, although in a
small range of differences, and were accompanied by the
cell surface expression of activation markers and
myeloperoxidase degranulation within granulocytes. The
post-prandial state, a physiological phenomenon that may
occur several times a day, could lead to a complex series of
events (including endotoxemia) that may initiate and develop
atherothrombosis.
The occurrence of similar predictive biomarkers in
conditions and diseases being apparently different for their
pathogenesis, such as vascular diseases and cancer, together