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Journal of General Virology (1995), 76, 2447-2456. Printed in GreatBritain 2447
Characterization, sequencing and phylogeny of the ecdysteroid
UDP-glucosyltransferase gene from two distinct nuclear polyhedrosis
viruses isolated from Choristoneurafumiferana
John W. Barrett, la Peter J. KrelF and Basil M. Arif l*
1Natural Resources Canada, Canadian Forest Service, Sault Ste. Marie, Ontario P6A 5M7
and 2Department of Microbiology, University of Guelph, Guelph, Ontario N1G 2 W1, Canada
The ecdysteroid UDP-glucosyltransferase (ego gene
isolated from a plaque-purified isolate of Choristoneura
fumiferana multinucleocapsid nuclear polyhedrosis virus
(CfMNPV) was compared to its homologue from a
defective MNPV virus (CfDEF) present in wild-type
virus populations infecting the eastern spruce budworm,
C. fumiferana. The egt genes were located in the same
relative position within the virus genomes and their
genomic location and arrangement were similar to that
found in Autographa californica MNPV (AcMNPV) and
Orgyia pseudotsugata MNPV (OpMNPV). The genes
encoded 491 and 494 amino acid open reading frames
respectively, and were 67 % identical at the amino acid
level and 74 % identical at the nucleotide level. Tran-
scripts of the egt of CfMNPV peaked around 12 h post-
infection (p.i.) and disappeared after 36 h p.i. Tran-
scripts of the egt of CfDEF peaked between 6 and 9 h p.i.
and were not detected 24 h p.i. The egt from CfMNP¥
was more similar to the partially sequenced egt identified
from OpMNPV, at the nucleotide and amino acid levels,
than it was to the egt from the CfDEF, AcMNPV,
Bombyx mori NPV, Lymantria dispar MNPV or Spodop-
tera exigua MNPV. Phylogenetic analysis of egt sup-
ported the baculovirus evolution scheme suggested by
polyhedrin sequence analysis.
Introduction
In insects, the moulting hormone ecdysone initiates and
regulates a cascade of genes expressed during moulting.
Recent studies have revealed that baculoviruses have
evolved mechanisms to delay normal lepidopteran larval
moulting. The ecdysteroid UDP-glucosyltransferase (egt)
gene, first identified in Autographa californica multi-
nucleocapsid nuclear polyhedrosis virus (AcMNPV),
catalyses the conjugation of ecdysone and UDP-glucose
(O'Reilly & Miller, 1989), and results in the clearing of
ecdysone from the haemolymph. The direct result is a
delay in moulting so that virus-infected larvae feed
longer and cause greater foliar damage. Interruption of
the AcMNPV egt coding region increased the virulence,
but did not reduce the infectivity of the virus. Insects
infected with the modified AcMNPV egt virus fed less
and died more rapidly than those infected with unaltered
* Author for correspondence. Fax + 1 705 759 5700. e-mail
barif@am.ncr.forestry.ca
The sequence data reported here will appear in the GenBank
Database under the accessionnumbers U10441 and U10476,
AcMNPV (O'Reilly & Miller, 1991). These advantages
suggested that alteration in the expression of egt in other
baculoviruses, either through deletion or inactivation,
might also provide a more effective biological control
insecticide.
The eastern spruce budworm, Choristoneura fumi-
ferana, is an economically important forest pest and is
responsible for the destruction of approximately 10"1
million hectares of forest per year in Canada (Anon-
ymous, 1994). To date, there is no effective and
economically viable biological control agent against this
devastating pest. At present, formulated Bacillus thu-
ringiensis (B.t.) is used to control forest lepidopteran pest
species. However B.t. has the potential to impact on non-
target lepidopteran species and has short persistence
times (Cunningham & van Frankenhuyzen, 1991).
Baculoviruses are naturally occurring pathogens which
are generally insect specific. It has been demonstrated
that genetic modification of baculoviruses can improve
their effectiveness as pest control agents (Wood &
Granados, 1991). Manipulation of the CfMNPV egt
appears to be one viable approach to improve baculo-
viruses against the spruce budworm. We report the
identification and characterization of the egt genes
0001-3271 © 1995SGM