Drug Development and Industrial Pharmacy, 2010; 36(11): 1330–1339
ISSN 0363-9045 print/ISSN 1520-5762 online © Informa UK, Ltd.
DOI: 10.3109/03639041003801885 http://www.informapharmascience.com/ddi
LDDI
ORIGINAL ARTICLE
Development of dorzolamide hydrochloride in situ
gel nanoemulsion for ocular delivery
Dorzolamide hydrochloride in situ gel nanoemulsion
H.O. Ammar
1
, H.A. Salama
1
, M. Ghorab
2
and A.A. Mahmoud
1
1
Department of Pharmaceutical Technology, National Research Centre Dokki, Cairo, Egypt and
2
Department of
Pharmaceutics, Faculty of Pharmacy, Cairo University, Cairo, Egypt
Abstract
Background: Several in situ gel-forming systems have been developed to prolong the precorneal resi-
dence time of a drug and to improve ocular bioavailability. Poloxamer 407 with its thermoreversible gela-
tion and surface active properties was utilized to formulate a novel dorzolamide hydrochloride in situ gel
nanoemulsion (NE) delivery system for ocular use. Objective: Improvement of both ocular bioavailability
and duration of action for dorzolamide hydrochloride was the aim of this study. Methods: Physicochemical
properties, in vitro drug release studies and biological evaluation of the prepared NEs were investigated.
Results: The optimum formulation of in situ gel NE consisted of Triacetin (7.80%), Poloxamer 407 (13.65%),
Poloxamer 188 (3.41%), Miranol C2M (4.55%), and water (70.59%). Biological evaluation of the designed
dorzolamide formulation on normotensive albino rabbits indicated that this formulation had better bio-
logical performance, faster onset of action, and prolonged effect relative to either drug solution or the
market product. The formula showed a superior pharmacodynamic activity compared to the in situ gel
dorzolamide eye drops. This indicated the effectiveness of the in situ gel properties of poloxamer 407,
besides formulating the drug in an NE form for improving the therapeutic efficacy of the drug. Conclusion:
These results demonstrate the superiority of in situ gel NE to conventional ocular eye drops and in situ
gels to enhance ocular drug bioavailability.
Key words: Bioavailability; dorzolamide hydrochloride; in situ gel; nanoemulsion; ocular
Introduction
Glaucoma is a serious eye disorder characterized by an
increase in intraocular pressure (IOP), which results in
damage to the optic disc
1
, and thus leads gradually to
loss of vision, usually without symptoms. Furthermore,
it is the second leading cause of blindness worldwide
2,3
.
It is believed that glaucoma is the result of an imbalance
between aqueous humor secretion and drainage pro-
cesses within the ocular chambers
4
.
Dorzolamide hydrochloride is a carbonic anhydrase
inhibitor used in the treatment of glaucoma. Carbonic
anhydrase inhibitors reduce IOP by decreasing aqueous
humor secretion through the inhibition of carbonic
anhydrase isoenzyme II in the ciliary processes
5
. Topi-
cally effective aqueous dorzolamide eye drop solution
(Trusopt
®
) has become one of the most widely used
medications for the treatment of open-angle glaucoma
since it became commercially available in 1995
6
. The
concentration of dorzolamide HCl in Trusopt
®
is 2.2%
(w/v), corresponding to 2.0% of the free base, pH 5.65.
Hydroxyethyl cellulose is used to increase the viscosity of
Trusopt
®
eye drops; this increased viscosity leads to
increased corneal contact time and, consequently, to
increased bioavailability. However, the relatively low pH
and high viscosity have been shown to generate local irri-
tation after topical administration of the eye drops
7
and
may be consistent with transient epithelial alteration
8
.
The anatomy, physiology, and biochemistry of the
eye render it highly impervious to foreign substances.
The absorption of drugs in the eye is severely limited by
some protective mechanisms that ensure the proper
functioning of the eye and by other concomitant
factors
9
. Several in situ gel-forming systems have been
Address for correspondence: Dr. H.O. Ammar, Department of Pharmaceutical Technology, National Research Centre, Dokki, Cairo, Egypt. Tel: +2 012
4472851, Fax: +2 02 33370931. E-mail: husseinammar@hotmail.com
(Received 24 Aug 2009; accepted 23 Mar 2010)
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