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Graft dysfunction in pancreas and islet transplantation:
morphological aspects
Cinthia B. Drachenberg and John C. Papadimitriou
Introduction
b-Cell replacement is the treatment of choice in a
subset of patients with diabetes mellitus. Insulin inde-
pendence can be achieved through two procedures:
whole pancreas transplantation (WPnTx) or islet trans-
plantation. Successful b-cell replacement immediately
improves the quality-of-life for the diabetic patient [1].
Furthermore, there is evidence that maintaining nor-
moglycemia leads to objective improvement of second-
ary diabetic lesions that were previously considered
irreversible [2]. The beneficial impact of pancreas
transplantation on secondary complications of diabetes
mellitus has been reviewed recently by Gremizzi et al.
[3
]. It is worth emphasizing that reversal of well
established diabetic lesions can be only achieved with
long-term normoglycemia. Specifically, reversal of
Kimmelstiel–Wilson nodules in diabetes nephropathy
was appreciated only in patients with WPnTx function-
ing at least10 years [2].
Although the first pancreas transplant was performed in
1966, achievement of acceptable results with WPnTx
has been slow, mostly due to a series of technical and
immunological difficulties [4]. Since the 1990s, results in
this area have continuously improved and WPnTx is
currently considered as a standard treatment for selected
patients in whom the surgical and immunosuppression
risks are outweighed by the morbidity and mortality
associated with their disease [5]. Long-term follow-up
of a cohort of 1000 patients receiving simultaneous
pancreas and kidney (SPK) transplantation showed that
at 1, 10 and 20 years, patient and pancreas graft survival
were 97, 80 and 58%, and 88, 63 and 36%, respectively [6]
The first series of successful islet transplantation were
reported in 2000, with achievement of excellent short-
term results (1 year insulin independence in 70 – 90% of
patients) [7]. Long-term results have been, however,
disappointing with return to insulin therapy within
5 years for most patients [8]. Despite the loss of insulin
independence over time, most patients with islet
Department of Pathology, University of Maryland
School of Medicine, Baltimore, Maryland, USA
Correspondence to Dr Cinthia B. Drachenberg, MD,
Department of Pathology, University of Maryland
Hospital, 22 South Greene Street, NBW49, Baltimore,
MD 21201, USA
E-mail: cdrac001@umaryland.edu
Current Opinion in Organ Transplantation 2011,
16:106–109
Purpose of review
b-Cell replacement in the form of whole pancreas transplantation (WPnTx) or islet
transplantation has the goal of providing long-term insulin independence to diabetic
patients that may require these types of interventions, with the minimum of iatrogenic
side-effects and complications. In search of these ambitious and only partially achieved
objectives, continuous advances are made in the field.
Recent findings
A concerted effort has been made in recent years to categorize the morphological
features of allograft rejection in WPnTx. This has followed the general attempts to
standardize histopathological and other diagnostic modalities in solid organ
transplantation in general. Issues related to antibody-mediated rejection have taken
center stage due to their perceived dramatic effects on both short and long-term graft
survival. Another issue that diminishes the extent of success with WPnTx is the high
incidence of posttransplant diabetes mellitus (PTDM). Understanding the mechanisms
involved in this process is important for the development of potential therapeutic
interventions and for its prevention.
Summary
This review will summarize the current understanding on the morphological features of
antibody-mediated rejection in WPnTx, the main morphological and clinical aspects of
PTDM, including recurrent autoimmune diabetes mellitus, and will briefly discuss
histopathological data available on islet transplantation.
Keywords
antibody-mediated allograft rejection, Banff grading schema, islet transplantation,
pancreas transplantation, post transplant diabetes mellitus
Curr Opin Organ Transplant 16:106–109
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1087-2418
1087-2418 ß 2011 Wolters Kluwer Health | Lippincott Williams & Wilkins DOI:10.1097/MOT.0b013e3283424f44