articles
NATURE CELL BIOLOGY VOL 3 DECEMBER 2001 http://cellbio.nature.com 1101
The GM130 and GRASP65 Golgi proteins
cycle through and define a subdomain
of the intermediate compartment
Pierfrancesco Marra*, Tania Maffucci*, Tiziana Daniele*, Giuseppe Di Tullio*, Yukio Ikehara†,
Edward K. L. Chan‡, Alberto Luini*, Gala Beznoussenko*, Alexander Mironov* &
Maria Antonietta De Matteis*§
*Department of Cell Biology and Oncology, Istituto di Ricerche Farmacologiche Mario Negri, Consorzio Mario Negri Sud, 66030 Santa Maria Imbaro (Chieti), Italy
†Department of Biochemistry, Fukuoka University School of Medicine, Fukuoka 814-0180, Japan
‡Department of Molecular and Experimental Medicine, The Scripps Research Institute, La Jolla, California 92037, USA
§e-mail: demattei@cmns.mnegri.it
Integrating the pleomorphic membranes of the intermediate compartment (IC) into the array of Golgi cisternae is a
crucial step in membrane transport, but it is poorly understood. To gain insight into this step, we investigated the
dynamics by which cis-Golgi matrix proteins such as GM130 and GRASP65 associate with, and incorporate, incom-
ing IC elements. We found that GM130 and GRASP65 cycle via membranous tubules between the Golgi complex and
a constellation of mobile structures that we call late IC stations. These stations are intermediate between the IC and
the cis-Golgi in terms of composition, and they receive cargo from earlier IC elements and deliver it to the Golgi
complex. Late IC elements are transient in nature and sensitive to fixatives; they are seen in only a fraction of fixed
cells, whereas they are always visible in living cells. Finally, late IC stations undergo homotypic fusion and establish
tubular connections between themselves and the Golgi. Overall, these features indicate that late IC stations mediate
the transition between IC elements and the cis-Golgi face.
T
he Golgi apparatus has a complex architecture composed of
stacks of flat cisternae connected in a ribbon-like fashion by
tubular reticular areas. How such an organization is achieved
at the cis-Golgi face and maintained through the Golgi in spite of
the continuous flux of membranes remains a central problem in
the biology of transport. Two classes of protein complexes are
thought to be involved in morphogenesis of the Golgi complex,
possibly through a coordinated action: the Golgi spectrin–actin
skeleton and the Golgi matrix proteins. The spectrin skeleton
assembles on Golgi membranes in a fashion dependent on the
activity of the small GTPase ARF (ADP-ribosylation factor) and on
the level of phosphatidylinositol 4,5-bisphosphate
1,2
. The spectrin
skeleton facilitates incorporation of endoplasmic reticulum (ER)-
derived membranes into the cis-Golgi
1
; it may also control the
structure of Golgi stacks, on the basis of the probable similarity of
the known role of spectrin at the plasma membrane
3
. The ‘Golgi
matrix’ proteins
4,5
were originally identified as a set of insoluble
Golgi proteins, and include GM130 and GRASP65. These proteins
are involved in postmitotic reassembly and stacking of the Golgi
cisternae
6–8
and can assemble in a structure reminiscent of the
Golgi even in the absence of Golgi resident proteins suggesting that
they can act as primary scaffold components underpinning the
Golgi architecture
9
. Remarkably, GM130, GRASP65 and other
Golgi matrix proteins are located primarily at the cis-pole of the
Golgi stacks
4,6
, where they probably function in the incorporation
of the ER-derived membranes into the Golgi.
The membranes that reach the cis-Golgi are tubular clusters that
constitute the ER–Golgi intermediate compartment (IC)
10
. The
biogenesis, maintainance, and identity of the IC have been and are
objects of intense study. Current theories suggest that the IC may be
considered either as an outgrowth of the ER
11,12
, as a part of the cis-
Golgi network (CGN)
13
, or as a separate compartment
14–17
. In the lat-
ter case, the IC might be viewed as a stable compartment
15,16
or as a
transient compartment made up of transport intermediates operat-
ing between the ER and the Golgi
14,17
. The difficulties in reconciling
these apparently contrasting views and in defining the boundary of
the IC result largely from the disperse distribution and pleomor-
phic organization of this compartment, and from its dynamic
behaviour
10
. Even the molecular composition of the IC elements is
highly heterogenous
18
and includes proteins continuously cycling
between the ER and the Golgi complex. However, by combining the
broadest definition of the ER–Golgi IC (consisting of the mem-
branes interposed between the ER and the Golgi stacks) with the
information derived from the dynamics of proteins traversing this
compartment (which includes cargo proteins
19,20
, recycling pro-
teins
21
, and the coat complexes COPI and COPII
22–24
), it is possible
to distinguish three layers in the IC. The first layer includes the ER
exit sites (ERES, or transitional ER), marked by COPII, and made
up of rather stationary elements
22,24
. The second layer consists of
tubular clusters that move long distances, travelling in centripetal
direction along microtubules; this layer contains COPI, but not
COPII, and forms what we define from here onwards as IC. The
third layer includes the CGN, which consists of tubular clusters jux-
taposed to the cis-Golgi cisterna (which in turn contain the cis-
Golgi markers GM130, GRASP65, mannosidase I, and Helix
Pomatia (HP) lectin-binding proteins
25
). According to the view of
the IC as a transient compartment, these three layers correspond to
three stages of the IC membrane lifespan: origin at the ERES via a
sorting process mediated by the SAR1p–COPII complex; motor-
driven translocation towards the Golgi along microtubules and
concomitant loss of ER-recycled components; and final incorpora-
tion into the Golgi complex. Although the first two stages have been
extensively studied and their molecular machineries satisfactorily
elucidated, the process by which the IC membranes are incorporat-
ed into the cis-Golgi remains elusive.
To gain insight into the details of this final step, we investigated
how Golgi-structural components such as the matrix proteins
GM130 and GRASP65 associate with the incoming IC membranes.
We find that IC elements acquire these Golgi components before
contacting the cis-pole of the central Golgi area, at the level of a
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