Research Article
Association of Levels of Antibodies from Patients with
Inflammatory Bowel Disease with Extracellular Proteins of
Food and Probiotic Bacteria
Arancha Hevia,
1
Patricia López,
2
Ana Suárez,
2
Claudine Jacquot,
3
María C. Urdaci,
3
Abelardo Margolles,
1
and Borja Sánchez
4
1
Instituto de Productos L´ acteos de Asturias, Consejo Superior de Investigaciones Cient´ ıficas (IPLA-CSIC),
Paseo R´ ıo Linares s/n, Villaviciosa, 33300 Asturias, Spain
2
Department of Functional Biology, Immunology Area, University of Oviedo, C/Juli´ an Claver´ ıa s/n, Oviedo, 33006 Asturias, Spain
3
UMR 5248 CBMN CNRS-Universit´ e Bordeaux 1-ENITAB, Laboratoire de Microbiologie et Biochimie Appliqu´ ee,
1 Cours du G´ en´ eral de Gaulle, 33175 Gradignan Cedex, France
4
Nutrition and Bromatology Group, Department of Analytical and Food Chemistry, Food Science and Technology Faculty,
University of Vigo, Ourense Campus, 32004 Ourense, Spain
Correspondence should be addressed to Borja S´ anchez; borja.sanchez@uvigo.es
Received 27 February 2014; Revised 18 May 2014; Accepted 19 May 2014; Published 4 June 2014
Academic Editor: Julio Villena
Copyright © 2014 Arancha Hevia et al. his is an open access article distributed under the Creative Commons Attribution License,
which permits unrestricted use, distribution, and reproduction in any medium, provided the original work is properly cited.
Inlammatory bowel disease (IBD) is an autoimmune disease characterized by a chronic inlammation of the gastrointestinal tract
mucosa and is related to an abnormal immune response to commensal bacteria. Our aim of the present work has been to explore the
levels of antibodies (IgG and IgA) raised against extracellular proteins produced by LAB and its association with IBD. We analyzed,
by Western-blot and ELISA, the presence of serum antibodies (IgA and IgG) developed against extracellular protein fractions
produced by diferent food bacteria from the genera Bifidobacterium and Lactobacillus. We used a sera collection consisting of
healthy individuals (HC, = 50), Crohn’s disease patients (CD, = 37), and ulcerative colitis patients (UC, = 15). Levels of IgA
antibodies developed against a cell-wall hydrolase from Lactobacillus casei subsp. rhamnosus GG (CWH) were signiicantly higher
in the IBD group ( < 0.002; = 52). he speciicity of our measurements was conirmed by measuring IgA antibodies developed
against the CWH peptide 365-VNTSNQTAAVSAS-377. IBD patients appeared to have diferent immune response to food bacteria.
his paper sets the basis for developing systems for early detection of IBD, based on the association of high levels of antibodies
developed against extracellular proteins from food and probiotic bacteria.
1. Introduction
Inlammatory bowel disease (IBD) is an autoimmune disease
characterized by a chronic inlammation of the gastroin-
testinal tract (GIT) mucosa. Depending on the severity and
location of the injuries, two main forms are distinguished,
Crohn’s disease (CD) and ulcerative colitis (UC). Both are
chronic disorders of unexplained origin, in which persistent
ulcerations appear in the small or large bowel mucosa.
Interestingly, genetic susceptibility only explains up to 23% of
the disease, in the case of CD (16% for UC), with the rest being
attributed to environmental factors, such as an exacerbated
response of the innate immune system to the commensal
microbiota [1].
Experiments in germ-free animals have shown that
microbial colonization is crucial in the instruction, matura-
tion, and regulation of the immune system. For instance, the
presence of Bacteroides fragilis ofers protection from experi-
mental colitis, induced by Helicobacter hepaticus, in an animal
model, with this beneicial activity being dependent on the
presence of an exopolysaccharide [2]. In addition, recent
metagenomic studies with human samples have revealed
that lifestyle in developing countries is associated with an
altered microbial colonization of the human gut [3]. Indeed,
Hindawi Publishing Corporation
BioMed Research International
Volume 2014, Article ID 351204, 8 pages
http://dx.doi.org/10.1155/2014/351204