175 Epigenomics (2015) 7(2), 175–186 ISSN 1750-1911
part of
Research Article
10.2217/EPI.14.77 © 2015 Future Medicine Ltd
Aims: We applied artificial neural networks (ANNs) to understand the connections
among polymorphisms of genes involved in folate metabolism, clinico-pathological
features and promoter methylation levels of MLH1, APC, CDKN2A
INK4A
, MGMT and
RASSF1A in 83 sporadic colorectal cancer (CRC) tissues, and to link dietary and
lifestyle factors with gene promoter methylation. Materials & Methods: Promoter
methylation was assessed by means of methylation-sensitive high-resolution melting
and genotyping by PCR-RFLP technique. Data were analyzed with the Auto Contractive
Map, a special kind of ANN able to define the strength of the association of each
variable with all the others and to visually show the map of the main connections.
Results: We observed a strong connection between the low methylation levels of
the five CRC genes and the MTR 2756AA genotype. Several other connections were
revealed, including those between dietary and lifestyle factors and the methylation
levels of CRC genes. Conclusion: ANNs revealed the complexity of the interconnections
among factors linked to DNA methylation in CRC.
Keywords: APC•artiicialneuralnetworks•CDKN2A•colorectalcancer
•DNAmethylation•folate•MGMT, MLH1,polymorphisms•RASSF1A
Colorectal cancer (CRC) evolves through a
stepwise accumulation of mutations and epi-
genetic modifications that transform normal
colonic cells into cancer [1,2] . Among epi-
genetic mechanisms, DNA methylation has
gained particular interest in cancer studies
because it was linked to the silencing of tumor
suppressor genes and DNA repair genes [3] .
Several genes are frequently hypermethylated
in sporadic CRC, including MLH1, APC,
CDKN2A, MGMT and RASSF1A [4–14] .
Common polymorphisms of folate meta-
bolic genes have been largely investigated
as CRC genetic risk factors, mainly because
folate metabolism (one-carbon metabolism)
in required for DNA synthesis and meth-
ylation (Figure 1), but literature data in this
field are conflicting and often insufficient to
clarify their contribution to DNA methyla-
tion and CRC risk [3] . This is likely due to
the complexity of this metabolic pathway
(Figure 1), and to the fact that traditional
statistical algorithms are often unsuitable to
dissect the relationship between high num-
ber of variables due to the nonlinearity and
complexity of their interactions [3] . In addi-
tion to genetic factors, dietary habits and
lifestyles, such as drinking and smoking, are
among the environmental factors suspected
to impair DNA methylation [15–17] .
In the present pilot study we applied Arti-
ficial Neural Networks (ANNs) to identify
genetic and dietary/lifestyle factors linked to
MLH1, APC, CDKN2A
INK4A
, MGMT and
RASSF1A promoter methylation in sporadic
CRC. ANNs aim to understand natural pro-
cesses and recreate those processes using auto-
mated models, and have been used success-
fully in gastroenterology and cancer studies
to understand nonlinear relationships among
variables [18–20] . Particularly, we applied the
Auto Contractive Map-Auto-CM algorithm
(Auto-CM), which is a peculiar ANN able
to define the strength of the associations of
Application of artificial neural networks to
link genetic and environmental factors to
DNA methylation in colorectal cancer
Fabio Coppedè*
,1,2,3
, Enzo
Grossi
4,5
, Angela Lopomo
1,6
,
Roberto Spisni
7
, Massimo
Buscema
5,8
& Lucia
Migliore
1,2,3
1
DepartmentofTranslationalResearch&
NewTechnologiesinMedicine&Surgery,
DivisionofMedicalGenetics,University
ofPisa,MedicalSchool,ViaRoma55,
56126Pisa,Italy
2
IstitutoToscanoTumori(ITT),
Florence,Italy
3
InterdepartmentalResearchCenter
Nutrafood‘Nutraceuticals&Foodfor
Health’,Pisa,Italy
4
BraccoFoundation,Milan,Italy
5
SemeionResearchCenter,Rome,Italy
6
DoctoralSchoolinGenetics,Oncology
&ClinicalMedicine,UniversityofSiena,
Siena,Italy
7
DepartmentofSurgery,Medical,
Molecular&CriticalAreaPathology,
UniversityofPisa,Pisa,Italy
8
DepartmentofMathematical
&StatisticalSciences,Universityof
ColoradoatDenver,CO,USA
*Authorforcorrespondence:
Tel.:+390502218544
fabio.coppede@med.unipi.it
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