Vol. 116, No. 3, 1983
November 15, 1983
BIOCHEMICAL AND BIOPHYSICAL RESEARCH COMMUNICATIONS
Pages 1049-1055
POSTTRANSLATIONAL MODIFICATION OF HUMAN
T-CELL GROWTH FACTOR
Richard J. Robb, RuSty M. Kutny, Maria Panico*, Howard Morris*,
William F. DeGrado, and Vinay Chowdhry
Central Research and Development Department,
E.I. du Pont de Nemours and Company, Wilmington, DE 19036
*Department of Biochemistry, Imperial College, London SW7, England
Received October 3, 1983
Amino-terminal sequence analysis of human T-cell growth factor
indicated that the amino acid in position 3 of the polypeptide
chain was modified. Examination of the N-terminal octapeptide
using the amino acid analyzer and mass spectrometry demonstrated
that position 3 was a threonine which was linked to N-acetyl-D-
galactosamine. This site of glycosylation is of practical
significance since it appears to play a role in the selectivity of
a monoclonal antibody for the factor.
T-cell growth factor (TCGF) I, also known as Interleukin
2, provides a signal for the proliferation of activated T-cells
during the T-cell immune response (1-3). The factor has been used
widely for the maintainance and cloning of non-transformed T-cells
in culture (2,4,5). In addition to its use in the laboratory,
several studies have indicated that defects in TCGF or its
production may be involved in certain pathological states (6-9).
A complete structural characterization of this lymphokine will
thus be valuable to an understanding of both the T-cell response
and the factor's potential clinical utility.
Using conventional biochemical
demonstrated that TCGF is a 15000MW
modification with a variable degree of
approaches, it was
protein subject to
glycosylation (I0).
Although there was no evidence that the carbohydrate affected in
vitro bioactivity, it was postulated that it might affect the
iT-cell growth factor, TCGF; phenylthiohydantoin, PTH; synthetic
decapeptide, Syn-P.
1049
0006-291X/83 $1.50
Copyright © 1983 by Academic Press, Inc.
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