ELSEVIER Psychiatry Research: Neuroimaging 67 (1996) 113-122
PSYCHIATRY
RESEARCH
NEUROIMAGENG
Multilead quantitative EEG profile of clozapine in resting and
vigilance-controlled conditions
Silvana Galderisi*, Armida Mucci, Paola Bucci, Maria Laura Mignone, Mario Maj
Department of Psychiatry, Medical School, University of Naples, Largo Madonna delle Grazie, 180138 Naples, Italy
Received 14 July 1995; revised 20 January 1996; accepted 28 January 1996
Abstract
Clozapine is the prototype of a new class of drugs, referred to as 'atypical antipsychotics'. As a matter of fact, the
antipsychotic activity of the drug was not predicted by the first studies with quantitative electroencephalography
(QEEG), which actually reported an antidepressant pattern. All previous QEEG studies carried out in healthy subjects
used a maximum of four leads, exploring only the posterior quadrants of the scalp. The present article reports findings
of a multilead QEEG study carded out in 16 healthy men under resting and vigilance-controlled conditions. Increases
in slow (delta, theta, and alpha1) and decreases in fast (alpha2 and beta) activities were found, corresponding to
changes described for chlorpromazine-type antipsychotics. These results are compared with those of earlier studies.
It is suggested that changes in the beta frequency range vary across subjects, whereas changes in slow and alpha activity
are more consistent and critical for defining the QEEG profile of the drug.
Keywords: Pharmaco-EEG; Atypical antipsychotics; Healthy subjects; QEEG mapping
1. Introduction
Clozapine is considered an atypical antipsy-
chotic, as it is devoid of cataleptogenic activity,
has a low propensity to produce extrapyramidal
side effects and tardive dyskinesia, and does not
increase prolactin levels in plasma (Baldessarini
and Frankenburg, 1991). In spite of the relatively
high risk of agranulocytosis, it is considered the
first-choice drug for patients with tardive
dyskinesia requiring antipsychotic treatment, and
* Corresponding author, Tel.: +39 81 5666504; fax: +39 81
5666523; E-mail: gsilvana@ds.cised.unina.it
for those who do not respond to standard
neuroleptics (Safferman et al., 1991). In these lat-
ter patients, clozapine has an impact on all
psychopathological dimensions of schizophrenia
(Kane et al., 1988; Meltzer, 1992; Pickar et al.,
1992; Breier et al., 1994).
Clozapine produces a weak antagonism of
striatal dopamine D2 receptors, and a more po-
tent blockade of DI, I)4, serotonin 5HT:, hista-
mine HI, Ctl, a2, and cholinergic receptors (Fitton
and Heel, 1990). In contrast to findings with
typical neuroleptics, the antipsychotic activity of
elozapine is not correlated to D2 receptor block-
ade and is thought to depend on its higher affinity
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