~) Pergamon
Int. J. Immunopharmac., Vol. 16, No. 10, pp. 835-844, 1994
Elsevier Science Ltd
Copyright © 1994 International Society for Immunopharmacology
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OHM3295: A FENTANYL-RELATED 4-HETEROANILIDO PIPERIDINE
WITH ANALGESIC EFFECTS BUT NOT SUPPRESSIVE EFFECTS ON
SPLENIC NK ACTIVITY IN MICE
DANIEL J. J. CARR,* MARC L. BAKER,* CHARLES HOLMES, t LINDA L. BROCKUNIER, t
JEROME R. BAGLEY ~ and CHARLES P. FRANCE t
Departments of *Microbiology, Immunology, Parasitology, and *Pharmacology, LSU Neuroscience Center,
Louisiana State University Medical Center, New Orleans, LA, 70112-1393, U.S.A.; and
*Ohmeda Inc., Murray Hill, New Jersey, U.S.A.
(Received 14 February 1994 and in final forrn 13 May 1994)
Abstract -- The immunoregulatory effects of fentanyl and a fentanyl-related compound, OHM3295, were
studied in mice. Male CD1 mice treated with a range of fentanyl doses (0.1 - 1.0 mg/kg, subcutaneously)
showed suppression of splenic natural killer (NK) activity following 0.25 - 0.50 mg/kg fentanyl dose but not
higher (0.75-1.0 mg/kg) or lower (0.1 mg/kg) doses. Fentanyl (0.01-32.0 mg/kg) also induced dose-
related analgesia as measured by an increase in tail flick latency; these analgesic effects were antagonized by
naltrexone (1.0-10.0 mg/kg). Pretreatment with naltrexone (1.0-3.2 mg/kg) resulted in significant
suppression of splenic NK activity following fentanyl (10.0 - 32.0 mg/kg) administration. In comparison to
fentanyl, OHM3~95 (3.2-25.0 mg/kg) augmented splenic NK activity in a naltrexone-reversible manner.
Similar to fentan3~l, OHM3295 (1.0- 32.0 mg/kg) also induced a naltrexone-sensitive, dose-related analgesia
as measured by an increase in tail flick latency. These results with OHM3295 demonstrate a novel profile of
effects which includes naltrexone-sensitive analgesic effects in the absence of immunosuppressive effects. In
addition, this is the first reported case in which a compound with opioid analgesic effects has been shown to
potentiate natural killer cytolytic activity following in vivo administration.
Morphine is a commonly used analgesic to control
acute and chronic pain; however, morphine and
heroin are potent immunosuppressive drugs in
rodents (Shavit etal., 1984; Bryant et al., 1987;
Pruett et al., 1992; Pacifici et aL, 1992; Bussiere
et aL, 1993; Freier & Fuchs, 1993), swine (Molitar
et al., 1992), non-human primates (Liu et aL, 1992;
Carr & France, 1993), and humans (Donahoe et aL,
1985; Novick etaL, 1989; Klimas etal., 1991).
Moreover, one manifestation of the immunosuppres-
sive effects of opioids in vivo is a diminished
resistance to bacterial (Tubaro et al., 1983) and viral
(Dismukes et al., 1986; Lorenzo et al., 1987)
infections.
Recently, the fentanyl derivative OHM3295 has
been shown to have opioid and non-opioid effects
which appear to be species specific. For example,
OHM3295 has been shown to displace 3H-naloxone
from/a-opioid binding sites in rat brain membranes
(Kd of 8.4 nM), to have analgesic effects in the
mouse hot-plate assay, and to reverse morphine-
induced respiratory depression in rabbits (Bagley
et al., 1991). In addition, OHM3295 can be substi-
tuted for naltrexone in drug discrimination tasks in
rhesus monkeys (France et al., 1994). However, in
monkeys the analgesic effects of OHM3295 are not
antagonized by naltrexone (France et al., 1994).
These opioid-like and non-opioid-like effects of
OHM3295 suggested that a further comparison of
this compound to typical /a-opioids might yield
additional novel effects. In the present study, the
immunoregulatory effects of OHM3295 and fentanyl
on splenic natural killer (NK) activity were assessed
concurrently with analgesic effects of these com-
pounds using the tail flick assay.
NK cells are a population of large granular
lymphocytes which have been associated with the
surveillance of viral infection and tumor growth and
metastases (Poste & Fidler, 1980; Herberman &
Ortaldo, 1981) through their ability to lyse infected
or neoplastic cells in a non-MHC-restricted fashion.
In rodents, the acute administration of morphine has
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