ELSEVIER Epilepsy Research 23 (1996) 71-76
EPILEPSY
RESEARCH
Effect of a high-protein meal on gabapentin pharmacokinetics
Barry E. Gidal a,b,*, Melissa M. Maly b, Jim Budde a, Gary L. Lensmeyer c,
Michael E. Pitterle a, John C. Jones b
a University of Wisconsin. School of Pharmacy. Madison. WI. USA
, University of Wisconsin. Department of Neurology. Madison. WI. USA
c University of Wisconsin Department of Laboratory Medicine. Madison. WI. USA
Received 17 April 1995; revised 15 July 1995; accepted 20 July 1995
Abstract
The anticonvulsant gabapentin is transported across biological membranes via the L-amino acid transport system
(System-L). Absorption of gabapentin is saturable, and in-vitro data have previously demonstrated that both L-leucine and
L-phenylalanine may compete with the intestinal transport of gabapentin. The purpose of this study therefore was to
determine whether a high-protein meal would interfere with gabapentin absorption. Ten healthy volunteers received in a
randomized, cross-over design, a single 600-mg dose of gabapentin in the fasting state and after a high-protein meal
consisting of 80 gm total protein (4.1 g phenylalanine, 8.2 g leucine and 4.2 g isoleucine), 52 g carbohydrate, and 9 g fat.
Plasma gabapentin concentrations were measured by HPLC at baseline, 0.25, 0.5, 0.75, 1, 1.5, 2, 2.5, 3, 3.5, 4, 5, 6, 8, 12,
24, 30 h. Calculated pharmacokinetic parameters included Cma x, Tma x, AUC and Tl/2. In addition, a pharmacodynamic
assessment (using visual analog scales) of gabapentin-related adverse effects was performed at 2 h post drug ingestion and
was compared between study phases. Statistical analysis included Student's t-test for paired data, with significance assigned
at P < 0.05. Cma ~ was significantly increased by 36% (3.87 -+ 1.15 vs 5.28 + .97/xg/ml, P = 0.002), and Tin, x tended to be
shorter (3.9 + 1.8 vs 2.8 + .35 h, P = 0.10), after the high-protein meal. Although AUC was increased by 1I%, this did not
achieve statistical significance. Despite significantly higher plasma concentrations at 2 h, subjects reported significantly
fewer adverse effects after the high-protein meal.
Potential mechanism:~ to explain these unexpected findings may be that the large amino acid load delivered with the
high-protein meal enhanced gabapentin absorption via trans-stimulation, the process by which acutely increased intestinal
luminal amino acid concentrations result in an acute up regulation in System-L activity. Conversely, the decrease in
perceived adverse CNS effects of gabapentin following the high-protein meal may reflect CNS competition for System-L
transport.
Keywords." Gabapentin;Pharmacokinetics;Drug-nutrient interaction; L-aminoacid transport system
1. Introduction
* Corresponding author. School of Pharmacy and Department
of Neurology, Unive~ity o1' Wisconsin-Madison,425 N. Charter
Street, Madison, WI 53706, USA. Tel.: (608) 262-3280; fax:
(608) 265-542I.
Gabapentin is a recently released anticonvulsant
medication that has demonstrated efficacy in the
treatment of complex partial seizures. Gabapentin is
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