Pergamon
0031-9384(94)00234-7
Physiology & Behavior, Vol. 57, No. 2, pp. 385-387, 1995
Copyright © 1995 Elsevier Science Ltd
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Lack of Sex and Estrous Cycle Effects
on the Activity of Three Antioxidant
Enzymes in Rats
VANIA D'ALMEIDA,* DI~BORA CRISTINA HIPrLIDEI"
AND MARIA EUGI~NIA DA SILVA-FERNANDES *l
Departments of *Biochemistry and ?Psychobiology, Escola Paulista de Medicina, S6o Paulo, Brasil
Received 14 March 1994
D'ALMEIDA, V., D. C. HIPOLIDE AND M. E. DA SILVA-FERNANDES. Lack of sex and estrous cycle effects on the activity
of three antioxidant enzymes in rats. PHYSIOL BEHAV 57(2) 385-387, 1995.--The activity of antioxidant enzymes was inves-
tigated in red blood cells of male and female Wistar rats 3-4 months of age. Superoxide dismutase (EC 1.15.1.1 ), catalase (EC
1.11.1.6), and glutathione peroxidase (EC 1. I 1.1.9) did not show any significant variation in the different phases of the estrous
cycle. No differences were observed for the three enzymes related to the sex of young rats. The present data enable us to consider
that sexual differences as well as the changes in estrous cycle do not interfere in erythrocyte antioxidant enzymatic defense of
rats.
Estrous cycle Sex differences Superoxide dismutase Catalase Glutathione peroxidase Free radicals
REACTIVE oxygen species (ROS) have been related to many
physiological and pathological processes. Organisms of an aer-
obic environment have developed a defense mechanism with a
group of essential enzymes: superoxide dismutase (SOD), cata-
lase (CAT), and glutathione peroxidase (GPx). The balance
among these enzymes and some nonenzymatic antioxidants (glu-
tathione, vitamin E, ascorbic acid, and other elements) is impor-
tant for the efficient removal of toxic oxygen from the intracel-
lular space.
Comparative studies of the activities of these enzymes have
shown variations between species (4,8) and tissues (1), but the
factors responsible for this variability are still unknown. Some
authors suggest that there are differences related to sex (2,5,6),
but the results are controversial.
Another possibility is the influence of sexual hormones on the
antioxidant defense (3,7,11,12). It is already known that vitamins
A, C, and E are present in corpus luteum and that their concen-
tration varies during the reproduction cycle (2). Evidences that
SOD may be regulated by luteinizing hormone have been found
(7,13). The analysis of SOD levels in thyroid of mice during the
estrous cycle showed differences that result in a peak of enzyme
activity in proestrus (13). These data led us to consider that fe-
male rats could show alterations in the enzymatic antioxidant
defense in the different estrous cycle stages.
To clarify these points, we studied the activity of antioxidant
enzymes (SOD, CAT, and GPx) in red blood cells (RBC) of male
and female rats 3-4 months of age. In the case of female rats,
we also determined the cycle stages for the evaluation of the
relationship between estrous cycle and antioxidant enzymatic de-
fense.
METHOD
Animals
The animals, 15 male and 39 female Wistar rats, were ob-
tained from the colony of the Department of Psychobiology of
Escola Paulista de Medicina, S~o Paulo, Brazil. The animals (3-
4 months old) were kept in groups of two or three in wire cages
with free access to food and water, and were maintained on a 12-
h light/dark cycle with lights on at 0700 h. The females were
divided in four groups according to their stage in the estrous cycle
at the moment of blood removal.
Determination of the Estrous Cycle Stages
Rats were monitored for normal estrous cycling by daily vag-
inal smears that were taken by a physiological saline lavage with
an eyedropper. The solution was examined under a microscope
for the cell content of the vaginal canal. Relative frequency of
leukocytes, cornified, and nucleated epithelial cells were noted.
Only female rats with a regular cycle served as subjects for this
study. To assure that females were cycling normally, all subjects
were initially monitored for 3 weeks to an adaptation period.
Requests for reprints should be addressed to Maria Eug~nia da Silva-Fernandes, Ph.D., Department of Biochemistry, Escola Paulista de Medicina,
Rua Botucatu, 862-Ed. Leal Prado -CP 20.372, Vila Clementino-04023-062, Sao Paulo, SP-Brasil.
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