Vol. 153, No. 1, 1988
May 31, 1988
BIOCHEMICAL AND BIOPHYSICAL RESEARCH COMMUNICATIONS
Pages 479-486
AJOENE INHIBITION OF PLATELET AGGREGATION : POSSIBLE
MEDIATION BY A H~OPROTEIN
Moideen P. Jamaluddin, Lissy K. Krishnan & Ancy Thomas
Thrombosis Research Unit, Sree Chitra Tirunal Institute for
Medical Sciences & Technology, Trivandrtun-695012, INDIA
Received April 29, 1988
SUMMARY. AJoene, an organosulfur compound derived from garlic,
was found by spectral measurements, to interact, cooperatively,
with a purified hemoprotein implicated, previously, in platelet
activation. It modified the binding interactions of the protein
with ligands, deemed to be physiologically relevant as effectors.
The characteristics of the modifications were found to parallel
those of aJoene induced modifications of agonist-induced aggreg-
ation kinetics of gel-filtered calf platelets. © z988 Aoademic P .....~nc
INTRODUCTION. AJ oene, (E, Z) -4, 5, 9-trithiadodeca-l, 6,11-triene
9-oxide, derived from garlic, irreversibly inhibits platelet
aggregation without affecting shape-change, metabolism of endo-
genous arachidonate, cAMP levels, and protein phosphorylation
(1-4). Recent evidence suggests its mechanism of action to be by
direct interaction with the fibrinogen receptor of platelets (4).
This mechanism, however, fails to explain the apparent cooperati-
vity of its inhibitory action (4)o Therefore an additional or
alternative site of action is suggested. We have implicated
ligand-induced conformational change of a dimeric hemoprotein in
platelet activation (5,6). Here we report on a parallelism found
between the modifying effects of aJoene on ligand binding to this
protein and the modifications it effected on the kinetics of
agonist-lnduced aggregation of calf platelets°
MATERIALS AND METHODS. AJoene was purified from freshly peeled,
crushed, garlic employing an adaptation of published procedures
(2). The purified compound was kept dissolved in 0.13 M NaC1, at
-30"C. H209 was a product of Glaxo (India). Other chemicals
were from c~mmercial sources given before (5,7-9). Gelfiltered
platelets (GFP) were prepared as described (7-9) except for the
following modifications : acid-citrate-dextrose solution replaced
trisodium citrate as anticoagulant (1.5 ml per 8.5 ml of blood)
479
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