Vol. 152, No. 3,1988
May 16,1988
BIOCHEMICAL AND BIOPHYSICALRESEARCH COMMUNICATIONS
Pages 95]-956
DEPLETION OF PROTEIN KINASE C INDUCED BY AN ANTI HLA CLASS I
MONOCLONAL ANTIBODY IN PHYTOHEMAGGLUTININ ACTIVATED
HUMAN T CELLS
Licia Poltronieri*, Edon Melloni**, Michele Rubini*,
Rita Selvatici*, Cristina Mazzilli***, Roberto Baricordi*
and Enrico Gandini*
*Istituto di Genetica Medica, Universit& degli Studi di Ferrara,
Ferrara, Italy
**Istituto Policattedra di Chimica Biologica, Universit~ degli
Studi di Genova, Genova, Italy
***Dipartimento di Biologia Sperimentale, Universit~ La Sapienza,
Roma, Italy
Received March 22, 1988
SUMMARY: Anti HLA Class I Monoclonal Antibody depletes Protein
Kinase C (PKC) to 20% of control value in PHA activated human T
cells. The effect is reversible: in 24 hours the enzymatic
activity returns to 58% of control value. Removal of antibody
from the culture medium increases the rate of recovery.
Implications of this finding for the modulation by HLA Class I
antigens of the proliferative response of T cells to lectins are
discussed. ® 1988 Academic Press, Inc.
Gene products encoded in the Major Histocompatibility
Complex (MHC) play a central role in immune response in man, as
in other animal species. Class I antigens are involved in
cytolytic T lymphocyte (CTL) response as shown for H2K/H2D
antigens in the mouse (i) and for HLA A and B antigens in human
(2,3,4,5), whereas MHC Class II antigens are largely involved in
antigen presentation and antigen specific interactions between T
cells and macrophages (6). Furthermore antibodies to MHC Class I
Abbreviations: PKC, protein kinase C; PHA, phytohemagglutinin;
FCS, fetal calf serum; MoAb, monoclonal antibody; PMA, phorbol
12-myristate 13-acetate; PMBC, peripheral mononuclear blood
cells; SRBC, sheep red blood cells.
951
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Copyright © 1988 by Academic Press, Inc:
All rights of reproduction in any form res#rved.