Original Paper
Ann Nutr Metab 2004;48:90–94
DOI: 10.1159/000077043
Annals of
Carnitine Supplementation Fails to
Maximize Fat Mass Loss Induced by
Endurance Training in Rats
Marcelo Saldanha Aoki
a,b
André Luı´s Rodriguez Amaral Almeida
a,d
Francisco Navarro
b,c
Luı´s Fernando Bicudo Pereira Costa-Rosa
a
Reury Frank Pereira Bacurau
b,c
a
Institute of Biomedical Sciences, USP, Sa ˜ o Paulo;
b
Laboratory Exercise Physiology – Faculty of Physical Education,
UniFMU, Sa ˜ o Paulo;
c
Lato Sensu Post-graduate Program – University Gama Filho, Rio de Janeiro, and
d
Faculty of Physical Education, UniSant’Anna, Sa ˜ o Paulo, Brazil
Received: March 31, 2003
Accepted: August 11, 2003
Published online: February 25, 2004
Marcelo Saldanha Aoki
Institute of Biomedical Sciences I
Av. Professor Lineu Prestes, 1524 s.410
University of Sa ˜o Paulo, Sa ˜o Paulo 05508-900 (Brazil)
Tel./Fax +55 3091 7311, E-Mail msaoki@usp.br
ABC
Fax + 41 61 306 12 34
E-Mail karger@karger.ch
www.karger.com
© 2004 S. Karger AG, Basel
0250–6807/04/0482–0090$21.00/0
Accessible online at:
www.karger.com/anm
Key Words
Carnitine supplementation W Endurance training W
Fat mass
Abstract
Background/Aims: Carnitine is a co-factor of the enzy-
matic system involved in long chain fatty acid transport
across the mitochondrial membrane. This physiological
role of carnitine raised the hypothesis that this com-
pound could act as a ‘fat burner’ by optimizing fat oxida-
tion and consequently reducing its availability for stor-
age. Our aim was to verify whether carnitine supplemen-
tation could maximize fat mass loss in trained rats. Meth-
ods: Male Wistar rats (200 g) were divided into four
groups: control (C), sedentary supplemented (S), trained
(T) and trained supplemented (TS). The training protocol
consisted of bouts of swimming exercise (60 min W day
–1
)
for 6 weeks. During the last 14 days, before sacrifice, the
supplemented groups received a daily dose of 28 mg W
kg
–1
of L-carnitine. Carcass fat content, weight and fat
content of adipose tissues were evaluated in all experi-
mental groups. Results: Our results indicate that carni-
tine feeding, per se, failed to promote fat mass loss.
Endurance training successfully induced a decrease in
the fat content in the carcass (28%) and the weight of
adipose tissues (retroperitoneal and mesenteric depots
by 41 and 20%, respectively) in comparison to C. Despite
the augmented carnitine content in the soleus mitochon-
dria (2-fold) observed in TS, the higher content did not
maximize the fat loss induced by endurance training.
Conclusions: Our data strongly suggest that endurance
training, rather than carnitine content, is the major factor
involved in fat mass loss.
Copyright © 2004 S. Karger AG, Basel
Introduction
It is well established that carnitine plays a key role in
lipid metabolism. Friedman and Fraenkel [1] demon-
strated that long chain fatty acid transport is a carnitine-
dependent process. The rate-limiting step in the ß-oxida-
tion of long chain fatty acids is assumed to be the trans-
port through the mitochondrial membranes [2, 3], which
is mediated by the carnitine palmitoyltransferase enzy-
matic system (CPT I, carnitine-acylcarnitine translocase
and CPT II) [4, 5]. The enhancement in CPT activity con-
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