IMAGES IN DERMATOLOGY Parwathi “Uma” Paniker, MD Rare Red Rash Shahid Hashmi, MD, Arash Heidari, MD, Arthur Jeng, MD Department of Medicine, Division of Infectious Diseases, Olive View-UCLA Medical Center, Sylmar, Calif. PRESENTATION Strolling through a farmers’ market can offer a pleasant way to spend an afternoon, but you may pick up more than a few fresh vegetables. Our patient’s symptoms began with a nonpruritic rash on his left shin, 4 weeks prior to presenta- tion. This seemingly harmless skin eruption quickly spread to bilateral lower and upper extremities, trunk, and abdo- men. A few days after the rash appeared, he developed fevers and night sweats. Upon presentation, the 34-year-old previously healthy Hispanic male had a 4-week history of fevers, night sweats, a 10-pound weight loss, and a wide- spread rash. Prior to the development of his symptoms, he was submerged in ocean water during his Jehovah’s Witness bap- tism ceremony and ate some cheese purchased at an open-air market. Working in a fast food restaurant, the patient has regular contact with food, but he denied any medication usage, animal exposure, insect bites, or recent travel. ASSESSMENT On physical exam, the patient appeared diaphoretic and was slow in answering questions. He had a temperature 38.7°C (101.7° F), pulse 119 beats/min, respiratory rate 19 breaths/ min, blood pressure 121/63 mm Hg. His physical examina- tion was significant for a palpable spleen and the dermato- logic findings of a papulonodular, violet-erythematous rash on the legs, arms, trunk, and abdomen, but not the palms and soles (Figures 1 and 2). On the legs, the rash was more confluent, with pitting edema extending to the knees. Laboratory results revealed white blood cell count 4.8 10 3 /L (63% neutrophils, 26% lymphocytes, 10% mono- cytes), hemoglobin 11gm/dL, platelets 140 10 3 /L, ESR 89 mm/hr, aspartate aminotransferase (AST) 104 units/L, alanine aminotransferase (ALT) 101 units/L, alkaline phos- phatase 153 units/L, total bilirubin 0.7 mg/dL. Other labo- ratory tests, including chemistry and urinalysis, were nor- mal. Serologic tests for HIV, rapid plasma reagin (RPR), hepatitis B and C, Coccidioides immitis, and anti-nuclear antibody (ANA) were negative. A transthoracic echocardio- gram was normal, with no vegetations. The chest radiograph was normal, and CT of abdomen/pelvis demonstrated mod- erate hepatomegaly and splenomegaly. Two sets of blood cultures were drawn on the day of admission. DIAGNOSIS The constellation of chronic fevers, mild hepatitis, hepato- splenomegaly, and maculopapular rash hinted at a several potential diagnoses. A lengthy list of possible causes for the patient’s symptoms was investigated: Rickettsial disease (Rickettsia typhi or murine typhus, which is endemic in southern California); Coxiella burnetti (Q fever), which can present with the rash as a consequence of subacute endocarditis; Bacterial endocarditis with immune-complex deposition; Fungal diseases such as coccidioidomycosis with ery- thema nodosum; Secondary syphilis; Typhoid fever with rose spots; Viral diseases including primary HIV and enteroviral infections; Vasculitidies. Many of the above diagnoses were ruled out with the blood tests and studies previously mentioned. After 3 days, the admission blood cultures signaled growth, and their gram stain revealed, unexpectedly, gram-negative coccoba- cilli that were speciated as Brucella melitensis (B.meliten- sis). Brucella serologies were positive at titers IgG1:2560 and IgM1:160. Brucellosis is a zoonotic infection caused by Brucella spe- cies, most commonly B. melitensis, found in sheep and goat, followed by B. abortis (found in cattle), and least commonly B. suis (swine) and B. canis (dogs). 1 Brucella is a small gram- negative, intracellular coccobacillus that is oxidase- and cata- lase-positive. This infection is commonly seen in Mediterra- nean, Asian, and Latin American countries. 2 In the US, it primarily occurs in Texas and California, where unpasteurized goat milk products imported from Mexico are its main source. 3 Requests for reprints should be addressed to Arthur Jeng, MD, Division of Infectious Diseases, Olive View UCLA Medical Center, UCLA School of Medicine, 14445 Olive View Dr. 2B182, Sylmar, CA 91342. E-mail address: artjeng@ucla.edu 0002-9343/$ -see front matter © 2008 Elsevier Inc. All rights reserved. doi:10.1016/j.amjmed.2008.07.005