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M.A. Hayat (ed.), Stem Cells and Cancer Stem Cells, Volume 8,
DOI 10.1007/978-94-007-4798-2_12, © Springer Science+Business Media Dordrecht 2012
12
Abstract
Embryonic stem (ES) cells have two defining
properties: self-renewal and pluripotency, and
these make them a promising source for cell
transplantation therapies. Oct4, Nanog, and
Sox2 are the main transcription factors in regu-
lating ES cell pluripotency. These key factors
have also been identified that form an intrinsic
core-regulatory circuit that maintains ES cells
in the pluripotent state in vitro. The precise
mechanism of how these processes are regulated
remains largely unknown. Thus investigation
of the molecular and cellular mechanisms of
stem cell self-renewal and pluripotency provide
the necessary tools to harness the regenerative
potential of ES cells for therapeutic purposes.
Recently, we have showed that natriuretic pep-
tide receptor A (NPR-A), a specific receptor for
atrial and brain natriuretic peptide (ANP and
BNP), is expressed in preimplantation embryos
and in ES cells, and is functional in ES cells.
In this chapter, we will provide an overview on
the importance of identifying the expression
and function of NPR-A in maintaining ES cell
characteristics.
Introduction
Embryonic stem (ES) cells are derived from inner
cell mass (ICM) of the blastocysts (Evans and
Kaufman 1981). They can undergo unlimited
self-renewal and characterized by their potential
Regulation of Self-Renewal
and Pluripotency of Embryonic
Stem Cells: Role of Natriuretic
Peptide Receptor A
Essam M. Abdelalim and Ikuo Tooyama
E.M. Abdelalim (*)
Department of Cytology and Histology, Faculty
of Veterinary Medicine, Suez Canal University,
Ismailia, Egypt
Molecular Neuroscience Research Center, Shiga
University of Medical Science, Setatsukinowa-Cho,
Otsu, Shiga 520-2192, Japan
e-mail: essam_abdelalim@yahoo.com
I. Tooyama
Molecular Neuroscience Research Center,
Shiga University of Medical Science,
Setatsukinowa-cho, Otsu, Shiga 520-2192, Japan
Contents
Introduction ............................................................ 123
Expression of NPR-A in Pluripotent ES
Cells and Pre-implantation Embryos ................... 124
Role of NPR-A in ES Cell Proliferation ............... 125
Role of NPR-A in ES Cell Pluripotency ............... 126
NPR-A Ligands Enhance
ES Cell Self-Renewal ............................................. 127
PI3K is Involved in NPR-A
Functions in ES Cells ............................................. 128
Differentiation of NPR-A-Deficient ES Cells ....... 128
References ............................................................... 130