The cytoplasm of every eukaryotic cell is elaborately
subdivided into discrete, specialized, membrane-bound
structures called organelles. Each organelle has a char-
acteristic morphology and is equipped with a specific set
of proteins and lipids to create a microenvironment that
is elegantly suited to carry out its defined functions in a
cell
1–4
. The different organelles communicate through
a constant exchange of material that, although enhancing
the metabolic efficiency of cells, does not compromise
each organelle’s steady-state composition and identity
5
.
The complexity in both the architecture and function of
organelles makes it energetically unfavourable for cells
to rapidly manufacture organelles de novo, even in the
case of organelles that originate from other intracellular
compartments
3,6
. Rather, a template-based biogenesis
mechanism involving the growth and division of pre-
existing organelles is the preferred method of maintain-
ing organelle populations during cell proliferation. With
each round of cell division, cells duplicate and apportion
their various organelles to the two resulting cells with
high accuracy, a process called organelle inheritance
1
.
In the past decade, considerable advances have
been made in understanding the molecular mecha-
nisms of organelle inheritance using the budding yeast
Saccharomyces cerevisiae. S. cerevisiae has facilitated
the study of organelle inheritance because its growth
is highly polarized, with a mother cell forming a bud
that is initially much smaller than itself. At first glance
it would seem that cells that divide by median fission
(for example, mammalian cells) need only to disperse
their organelles randomly in the cytoplasm to achieve
organelle inheritance on cytokinesis; however, organelle
partitioning in these cells has also been shown to be an
ordered process involving the cytoskeleton and motor
proteins
7–12
. This Review highlights the recent progress
made in uncovering the molecular basis of perox-
isome inheritance in yeast; however, we do not hesitate
to diverge from the field of peroxisome inheritance to
draw the reader’s attention to fascinating complemen-
tary findings arising from studies of inheritance of
other organelles. One emergent theme is that, although
each organelle uses specific molecular components to
ensure its inheritance by future generations of cells, a
set of fundamental rules applies to the mechanisms of
inheritance of all organelles. The timing of this Review
coincides with an unprecedented understanding of these
common denominators, leading to the formulation of
unifying themes and testable general paradigms for the
partitioning of all organelles.
Organelle inheritance in budding yeast
S. cerevisiae multiplies by a repetitive pattern of growth
and division termed budding. At the beginning of each
cell cycle, cells select a site for bud emergence based
on physical cues from previous cell cycles
13,14
. Among
the signalling molecules and polarity-establishing
factors attracted to this future bud site is a conserved
class of proteins called formins
14,15
. Formins function in
assembling unbranched actin filaments by holding on
to the plus end of an actin filament while catalysing the
incorporation of new actin monomers
16–19
. As formins
are strategically positioned at the future bud site, they
Department of Cell Biology,
University of Alberta, Medical
Sciences Building 5-14,
Edmonton, Alberta, T6G 2H7,
Canada.
Correspondence to R.A.R
e-mail: rick.rachubinski@
ualberta.ca
doi:10.1038/nrm2960
Published online
18 August 2010
Formin
One of a group of conserved
proteins that nucleate actin
assembly by promoting the
incorporation of new actin
monomers into the growing
plus end of an actin filament,
with which they remain
associated.
Actin monomer
A monomer of actin (also
known as globular actin
(G-actin)) that polymerizes into
helical actin filaments called
filamentous actin (F-actin) or
microfilaments.
Molecular mechanisms of organelle
inheritance: lessons from peroxisomes
in yeast
Andrei Fagarasanu, Fred D. Mast, Barbara Knoblach and Richard A. Rachubinski
Abstract | Preserving a functional set of cytoplasmic organelles in a eukaryotic cell requires a
process of accurate organelle inheritance at cell division. Studies of peroxisome inheritance
in yeast have revealed that polarized transport of a subset of peroxisomes to the emergent
daughter cell is balanced by retention mechanisms operating in both mother cell and bud
to achieve an equitable distribution of peroxisomes between them. It is becoming apparent
that some common mechanistic principles apply to the inheritance of all organelles, but
at the same time, inheritance factors specific for each organelle type allow the cell to
differentially and specifically control the inheritance of its different organelle populations.
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