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Geometry as a Factor for Tissue Growth: Towards Shape
Optimization of Tissue Engineering Scaffolds
Cécile M. Bidan, Krishna P. Kommareddy, Monika Rumpler, Philip Kollmannsberger,
Peter Fratzl,* and John W. C. Dunlop
Scaffolds for tissue engineering are usually designed to support cell viability
with large adhesion surfaces and high permeability to nutrients and oxygen.
Recent experiments support the idea that, in addition to surface roughness,
elasticity and chemistry, the macroscopic geometry of the substrate also con-
tributes to control the kinetics of tissue deposition. In this study, a previously
proposed model for the behavior of osteoblasts on curved surfaces is used to
predict the growth of bone matrix tissue in pores of different shapes. These
predictions are compared to in vitro experiments with MC3T3-E1 pre-osteob-
last cells cultivated in two-millimeter thick hydroxyapatite plates containing
prismatic pores with square- or cross-shaped sections. The amount and
shape of the tissue formed in the pores measured by phase contrast micro-
scopy confirms the predictions of the model. In cross-shaped pores, the ini-
tial overall tissue deposition is twice as fast as in square-shaped pores. These
results suggest that the optimization of pore shapes may improve the speed
of ingrowth of bone tissue into porous scaffolds.
of cell and tissue responses and to design
optimal scaffolds for in vivo experiments
and applications.
Cells are known to adapt to the phys-
ical properties of their surroundings by
integrating the mechanical equilibrium
established at their adhesion sites.
[5]
The
resulting mechanical cue is translated
into a biochemical signal that triggers bio-
logical decisions of the cells.
[6]
As cells are
mechanically attached to each other, either
directly or via their extracellular matrix,
they are also able to synchronize their
response on a larger scale. For example,
patterning in cell differentiation arises as
a response to stiffness
[7]
or strain
[8]
pat-
terns, and the distribution of proliferation
activity also correlates with the stress dis-
tribution in a layer of cells.
[9]
Cell fate has also been investigated
in three-dimensional artificial scaffolds.
Adhesion, proliferation, differentiation and mineralization of
cells and tissues have been compared in several scaffolds with
varying structures.
[10,11]
Recently, Kumar et al.
[12]
showed that
gene expression, and thus cell differentiation, is more affected
by the structural properties of the substrate than by its com-
position. Furthermore, pore size and porosity need to satisfy
the compromise between a high permeability that enables
cell migration and nutrient diffusion within the scaffold, and
a large surface area for cell adhesion and extracellular matrix
production.
[3]
Many fabrication processes produce structures
with random pores in a large range of sizes and interconnec-
tivities difficult to control. Rapid prototyping techniques are
much more accurate in that respect.
[13]
The direct printing of
the scaffold enables to control the architecture and thus many
mechanical properties of the structure.
Rumpler et al.
[14]
used rapid prototyping to build artificial
macro-pores of different controlled geometries and showed that
cells locally respond to high curvature by producing tissue. Their
hypothesis of local tissue growth proportional to curvature has
been confirmed experimentally, not only in pores but also on
open surfaces,
[15]
however with the additional observation that
tissue does not grow on convexities. The interfacial evolution
derived from a curvature-driven tissue growth model matched
the experimental observations as well as the in vivo expecta-
tions when comparing with the typical geometries involved in
1. Introduction
Three-dimensional scaffolds are needed for tissue engineering
applications and may also help to study the effect of the environ-
ment on tissue growth in vitro. The material used,
[1]
the fabrica-
tion process,
[2]
and the architecture of the scaffold
[3,4]
are known
to influence the biological interactions with the host organism.
Although all these parameters are difficult to decouple, quanti-
fying their effects in vitro is necessary to understand the nature
DOI: 10.1002/adhm.201200159
C. M. Bidan, Dr. K. P. Kommareddy,
Dr. P. Kollmannsberger, Prof. P. Fratzl,
Dr. J. W. C. Dunlop
Department of Biomaterials
Max Planck Institute of Colloids and Interfaces
14424 Potsdam, Germany
E-mail: peter.fratzl@mpikg.mpg.de
Dr. M. Rumpler
Ludwig Boltzmann Institute of Osteology at the Hanusch Hospital
of WGKK and AUVA Trauma Centre Meidling
1th Medical Department
Hanusch Hospital, Vienna, Austria
Dr. P. Kollmannsberger
Department of Health Sciences and Technology (D-HEST)
ETH Zurich, 8093 Zurich, Switzerland
Adv. Healthcare Mater. 2013, 2, 186–194