Copyright © 2007 Undersea and Hyperbaric Medical Society, Inc 407
UHM 2007 Vol.34, No.6 – Apnea in underwater hockey players
Physiological responses to repeated apneas
in underwater hockey players and controls.
Submitted 5/3/07; Accepted 6/20/07
F. LEMAÎTRE
1,2
, D. POLIN
3
, F. JOULIA
4,2
, A. BOUTRY
3
, D. LE PESSOT
3
, D. CHOLLET
1
,
C.TOURNY-CHOLLET
1
1
Centre d’Etudes des Transformations des Activités Physiques et Sportives, Equipe d’Accueil UPRES N°3832. Faculté des Sciences
du Sport et de l’Education Physique de Rouen, Université de Rouen, France,
2
Association pour la Promotion de la Recherche sur
l’Apnée et les Activités Subaquatiques (A.P.R.A.A.S.),
3
Service de Physiologie Respiratoire et Sportive, Hôpital de Bois Guillaume,
76031 Rouen,
4
Laboratoire d’Ergonomie Sportive et Performance Motrice ESP, UPRES EA n°3162, UFRSTAPS de Toulon,
Université de Toulon et du Var, BP 132, 83957 La Garde, France.
Lemaitre F, Polin D, Foulia F, Boutry A, LePessot D, Chollet D, Tourny-Chollet C. Physiological responses
to repeated apneas in underwater hockey players and controls. Undersea Hyperb Med 2007; 34(6):407-
414. The aim of this study was to investigate the effects of short repeated apneas on breathing pattern and
circulatory response in trained (underwater hockey players: UHP) and untrained (controls: CTL) subjects.
The subjects performed five apneas (A1-A5) while cycling with the face immersed in thermoneutral water.
Respiratory parameters were recorded 1 minute before and after each apnea and venous blood samples were
collected before each apnea and at 0, 2, 5 and 10 minutes after the last apnea. Arterial saturation (SaO
2
) and
heart rate were continuously recorded during the experiment. Before the repeated apneas, UHP had lower
ventilation, higher P
ET
CO
2
(p<0.05) and lower P
ET
O
2
than CTL (p<0.001). After the apneas, the P
ET
O
2
values
were always lower in UHP (p<0.001) than CTL but with no difference for averaged P
ET
CO
2
(p=0.32). The
apnea response, i.e., bradycardia and increased mean arterial blood pressure, was observed and it remained
unchanged throughout the series in the two groups. The SaO
2
decreased in both groups during each apnea
but the post-exercise SaO
2
values were higher in UHP after A2 to A5 than in CTL (p<0.01). The post-
apnea lactate concentrations were lower in UHP than in CTL. These results indicate that more pronounced
bradycardia could lead to less oxygen desaturation during repeated apneas in UHP. The UHP show a specific
hypoventilatory pattern after repeated apneas, as well as a more pronounced cardiovascular response than
CTL. They indeed showed no detraining of the diving response.
INTRODUCTION
The diving response is characterized
by bradycardia, peripheral vasoconstriction,
decreased cardiac output, and a gradual
elevation in arterial pressure with consequent
lactate accumulation in unperfused muscle, and
elite apnea divers display a more pronounced
response than novices (1). Apnea diving is a
new sport that is usually characterized by
prolonged apneas at rest or at low exercise
intensity. However, in several sports, such as
judo, track and field, basketball and soccer,
athletes are required to perform high intensity
intermittent exercise. Intermittent training
decreases the recovery period after submaximal
exercise (2) and this effect has been explained
by a smaller increase in lactate concentration,
an improvement in the quantity of oxygen
available to active muscles, and better oxygen
extraction (3). Underwater hockey players
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